mormiles wrote:> MS which is universally accepted as a chronic inflammatory disease.
Everyones's MS is different, but it's all inflammatory.
historically, several different treatments have enjoyed some limited success that never proved to be either lastiing or univeral in effectiveness (gluten/casein free diet, other allergy treatments, avoiding excitotoxins, chelation, antifungals, etc.)
Anecdote wrote:The excuses for not having done a test with antibiotics is by no means lame. The last test that was tried at Vanderbilt too many people dropped out for whatever reason. My view is that it is not fair to do a double blind trial when you could just be benefitting from the treatment. I couldn't have waited two years and then found that I was on the placebo arm: I wanted to get better. Doesn't everyone? Obviously not.
Anecdote wrote:Who do you mean when referring to "we?"
The people at Vanderbilt, including the head neurologist, Ram Sriram, want nothing more than to help patients. However, patients do not tend to want to feel worse when they start on a trial. Antibiotics tend to make you feel worse before you start to feel better. The people who don't realize this drop out. If the trial started with only twenty people or less numbers can soon drop below the required amount. You can't force anyone to do something that they don't want to.
Anecdote wrote:I said that there have been two trials at Vanderbilt but the second one closed because too many people dropped out. In my country, the UK, trials are well nigh impossible to organise for MS unless you have a neurologist involved but the current batch of neuros believe too much in the auto-immune theory: at the moment anyway. Maybe you could organise something in Serbia? Then the UK might suddenly show more interest.
I maybe should have said that antibiotics which are working tend to make someone feel worse for a few hours or the first day or two, depending on how fast their metabolism is.
My husband is a doctor, a consultant microbiologist who knows a good deal about the bactreria found in people’s bowels and elsewhere and is fully aware about chronic diseases and gut flora. It was him, after all, who wanted e to start this regime after reading th Vanderbilt patent. I was the one who was very doubtful until it started to work.
Anonymoose wrote:I am following the Wheldon protocol and have found that mcp helps immensely with the resulting inflammation. I've done a little research on mcp and suspect that it's ability to bind to inflammation causing galectin-3 (present in elevated levels in ms and arteriosclerosis plaques) is what makes it so helpful to me. It also acts as a gentle chelator. It might be of use to pwms even if they aren't on an antibiotic protocol so I thought I'd share.
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