drsclafani wrote:
my own sense is that cranial nerve and hypothalamic compression is more likely the culprit here. Increases in cerebrospinal pressure probably related to venous outflow reduction's negative effect on cerebrospinal flud drainage causes some minimal increases in pressure in the CSF and downward movement of the brainstem toward the foramen magnum. I think that the compression of the brain stem at the foramen magnum is subtle but may be significant. I certainly have an easier time accepting nearly instantaneous improvements explained by improved CSF drainage than effects on the Vagus nerve.
Not definitive but my sense of things.
S
Cranial nerve compression (or relief of) is a logical explanation for the instantaneous eyesight improvements reported in many patients (including my wife).
As a result of your post above it prompted me to read as much as I could about CSF and it's flow. Which, as usual has left me with far more questions than answers.
This is where Dr Amir's theories start to coincide with your possible theories about the importance of CSF flow.
Do we currently (medically speaking) completely understand the flow mechanisms of CSF? The reading I've done this evening seemed to contain lots of "Mays" and "Possiblies" when discussing routes of flow for CSF.
The following is a cut and past from that oracle of the interweb, Wiki, explaining in language I mostly understand what CSF is and what it does.
CSF serves four primary purposes:
Buoyancy: The actual mass of the human brain is about 1400 grams; however, the net weight of the brain suspended in the CSF is equivalent to a mass of 25 grams. The brain therefore exists in neutral buoyancy, which allows the brain to maintain its density without being impaired by its own weight, which would cut off blood supply and kill neurons in the lower sections without CSF.
Protection: CSF protects the brain tissue from injury when jolted or hit. In certain situations such as auto accidents or sports injuries, the CSF cannot protect the brain from forced contact with the skull case, causing hemorrhaging, brain damage, and sometimes death.
Chemical stability: CSF flows throughout the inner ventricular system in the brain and is absorbed back into the bloodstream, rinsing the metabolic waste from the central nervous system through the blood–brain barrier. This allows for homeostatic regulation of the distribution of neuroendocrine factors, to which slight changes can cause problems or damage to the nervous system. For example, high glycine concentration disrupts temperature and blood pressure control, and high CSF pH causes dizziness and syncope. To use Davson's term, the CSF has a "sink action" by which the various substances formed in the nervous tissue during its metabolic activity diffuse rapidly into the CSF and are thus removed into the bloodstream as CSF is absorbed.
Prevention of brain ischemia: The prevention of brain ischemia is made by decreasing the amount of CSF in the limited space inside the skull. This decreases total intracranial pressure and facilitates blood perfusion.Questions:-
Is it possible that treating CCSVI is actually about promoting better CSF flow rather than just bloodflow?
Is it possible that skeletal misalignment correction (atlas correction etc) is promoting better CSF flow?
This would explain at least some of what appear to be similar improvements generated by these different approaches.
Then logically, getting your skeletal alignment straight and being treated for CCSVI might be required in some, but not all patients?
Or have I put 2 & 2 together and got 10 again? Which is something I seem to have a habit of doing.
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