Vascular formation and stent types

A forum to discuss Chronic Cerebrospinal Venous Insufficiency and its relationship to Multiple Sclerosis.

Vascular formation and stent types

Postby Shannon » Mon Apr 05, 2010 5:18 am

Hi everyone - I was just looking at some research this morning and I was wondering if this is interesting to anyone:

http://www.sciencedaily.com/videos/2008 ... stents.htm

I am not familiar with the types of stents that have been used already, but these look to be very promising! The article is from 2008.

Also, here is the article that got me started reading, and there are also related articles on this page to read about vascular development. I think it really supports congenital blood vessel growth in CCSVI. I think we all have a variety of collateral blood vessels that have formed to make up for the jugular vein malformation, and for some of us these new vessels became compromised somehow. In my case, I was almost strangled. I bet the older we get, the new blood vesel formation becomes slower and less likely. Enjoy some early morning reading! :

http://www.sciencedaily.com/releases/20 ... 7101416365
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Some Related New Research

Postby Squeakycat » Mon Apr 05, 2010 6:26 am

Not that everything "vascular" is relevant, this seemed important in understanding the development of collateral veins.


Focusing on the development of the fifth and sixth aortic arches in the zebrafish, Dr. Lawson describes how the forces exerted by blood flow on endothelial cells are a critical component for expressing a microRNA that triggers new vessel development. In the early stages of development, when blood flow is present in the aortic vessels, but the vascular linkages between the two arches have yet to be established, the stimulus provided by active blood flow leads to expression of an endothelial-cell specific microRNA (mir-126). In turn, this microRNA turns on vascular endothelial growth factor (VEGF), a chemical signal produced by surrounding cells that normally stimulates angiogenesis. Thus, blood flow allows the endothelial cells to respond to VEGF by growing into new blood vessels. However, when blood flow in the aortic arches was restricted, mir-126 failed to be expressed. In the absence of this microRNA, new blood vessels were unable to develop due to a block in VEGF signaling.


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