It's been frustrating to see neurologists call my husband's improvements of fatigue, cognitive fog and heat tolerance "placebo", when shunted NPH patients results are seen as medical fact. Jeff has a stent high in his left tranverse sinus. Since its placement, he no longer has tinnitus or headaches. This endovascular treatment is approved for those with intracranial hypertension, tinnitus, and papilledema. Jeff had all of these symtoms since childhood, but got an MS diagnosis due to the numerous white matter lesions at his first MRI at age 44. As Dr. Flanagan says, these white matter hyperintensities are seen in ischemia, migraine, and traumatic brain injury with edema. And Dr. Frohman admitted he'd seen white matter lesions in his NPH patients, after they were shunted. It was the enlarged third ventricle which covered up the white matter lesions, and when the CSF was diverted, the ventricle shrank and the white matter lesions became evident. Here's Dr. Frohman's quote from 2009.
I have seen this happen in “normal pressure hydrocephalus- (NPH) Where there is a loss of gait, cognitive and bladder issues and the lesions disappear because the expanded ventricle swallows the lesion. I have shunted the brains of NPH patients, and they showed remarkable improvements. Again, the enlargement of the third ventricle precedes the changes.
and here's the note I wrote up about NPH and the discovery of Dr. Hakim, made in the 1960s--- for those on Facebook.
https://www.facebook.com/notes/ccsvi-in ... 7640167211
Thanks to all who are discussing the importance of CSF and venous return in MS, and thanks to Dr. Flanagan for his clear and brilliant observations on how the body's architecture plays an important part. I hope it brings answers for more people. We're getting there!