Thanks Joan I have ordered Doc Cooke's book and waiting.
I always look at things as WHY and the reason for high "Endothelin-1, a smooth muscle mitogen" occurring has my total attention.
It is interesting that there hasn't been further research looking at the incidence and coming to an understanding.
The other aspect is that there is a stand off between researchers regarding the causation of arteriosclerosis, one camp saying it is bacterially caused and the other flapping in the wind saying no it isn't! Yet the research is showing that treating with ABx protocols stops the progression of it! So there seems to be the same division in research across the board and that is why the Stealth Infection work of Prof Nicolson and others is falling on deaf ears. Bec Miller is providing a big push across the ditch in Australia regarding this and it has my attention as well. Many other Specialists that Bec has interviewed are connecting the dots Internationally. So what I am saying is that the diseases implicated eg atherosclerosis and also RA are also on the list for CPn infection incidence which for me is too 'interesting' to pass by.
I'm not saying that the 'standard' diseases implicated 'are the reason for MS/CCSVI/de-generative diseases', what I do see is that they play an active role in the jigsaw, and so does your list and the injury and spinal alignment collective. Some of us like me have the lot and have this thing called MS! A singular approach to treating only one of the factors is not going to protect against the disease as you have found with Jeff's multifaceted treatment program.
For me I have a raft of things to attend to and also keep an open mind as I hear about the scenarios of others, such as Marie and Mark Stecker who have been able to cross off some of the package or list of co-incidents as technology proves 'beyond doubt' that these issues eg Bacterial Infection are not an issue, or is it?
I am of course challenged deeply that technology isn't able to give better than a 60% reliable antigen test for Lyme or CPn involvement, so are Marie and Marc clear, or are they in a situation where the previous 'drugs' and Lifestyle/diet used enabled the bacteria to adapt their DNA and become even more of a Stealth risk in the equation? Monocytes and Marcophages still being hosts to the intracellular bacteria and spreading the bacterial Farmlets around the body still?
And that is why I look at this Endothelin-1, a smooth muscle mitogen as a new incidence factor to understand in depth and from all perspectives.