This is shoshin's blog, she is one of the Dake originals. This is from December 2010, so just a few months ago.
Interestingly, Dr. Dake said they saw a similar course to mine in five or six other people out of the 40 or so they treated. I got the impression these were more progressive patients, but now I'm not sure. People in this category saw an improvement, sustained or mostly sustained, in what they are calling global symptoms, such as fatigue, cog fog, and temperature sensitivity, which Dr. Dake thinks are the sort of things most likely to be helped by improved blood flow. These patients also saw improvements in motor and other symptoms where it is less obvious how improved blood flow would be effective. As in my case, after some time, these results wore off and they began to worsen. The people at Stanford don't know why this might have happened.
What do we make of this? Initial or maybe sustained improvement in all the "cerebral MS" stuff, even in the motor areas initially, but then continued decline in the motor areas?
Could it be missed azygous stenosis? Or MS having an autoimmune component, that has been triggered by the immune system's access to the brain and can't be stopped just by fixing the vascular stenoses? Could it be restenosis?