Perhaps you might share the titles of some of those papers.
You give ONE paper on ONE healthy patient followed for only 4 years, patientx....that's not exactly conclusive evidence.
In the present study, we demonstrated that the healthy brain can tolerate bilateral jugular vein ligation during radical neck dissection by opening invisible channels.
My husband's brain was anything but healthy when he was treated for severe bilateral jugular stenosis. And he had it for over 30 years, since he lost his peripheral eyesight at age 10. Pseudotumor cerebri (what he had) is linked to jugular vein ligation or blockage.
here you go:
Titles and authors of 14 papers showing a variety of effects of jugular ligation on the brain on numerous patients- including change in evoked potentials, interference with reabsorption of CSF, ischemia, blindness, optic neuropathy and edema and 8 deaths. Not exactly benign.
Serious cerebral complications following unilateral ligation of the internal jugular vein. Review of the literature and a case report.
Rolffs J, Schmelzle R.
Ophthalmoplegia secondary to raised intracranial pressure after unilateral neck dissection with internal jugular vein sacrifice
Matthew J. A. Ward MRCS1,*, Callum Faris MRCS1, Tahwinder Upile FRCS2, Nimesh N. Patel FRCS1
YungMW, SolimanAM. Changes in the brainstem evoked responses following jugular vein ligation. J Laryngol Otol 1988;102:861-864
The effect of internal jugular vein ligation on cerebral circulation in children with extrahepatic portal hypertension
[Article in Russian]
Piroganov FF, Razumovskiĭ AI, Gorbachëv OS, Stroganova TS, Ustinova OB.
KawajiriH, FuruseM, NambaR, KotaniJ, OkaT. Effect of intemal jugular vein ligation on resorption of cerebrospinal fluid. J Maxillofac Surg 1983;11:42-45
HasanM, SrimalRC, MaitraSC .Bilateral jugular vein ligation- induced alterations in the ventricular ependyma: Scanning electron microscopy. Int Surg 1980;65:533-540
ANTERIOR ISCHEMIC OPTIC NEUROPATHY FOLLOWING NECK DISSECTION
Scott E. Strome, MD,1 Jimmy S. Hill, MD,1 Michael A. Burnstine, MD,2 Jill Beck, MD,1 Douglas B. Chepeha, MD,1 Ramon M. Esclamado, MD3
Blindness following bilateral radical neck dissection
Steven C. Marks MD1, Dr. Darrell A. Jaques MD2,3,*, Richard M. Hirata MD3, John R. Saunders Jr. MD3
YuraS,SakoK,YonemasuY.The effect of disturbance of cerebral venous drainage on focalc erebral bloodflow and ischemic cerebral edema [inJapanese].Noushinkei 1990;42:269-275
Royster HP. The relationship between internal jugular vein pressure and cerebrospinal fluid pressure in the operation of radical neck dissection. Ann Surg 1953;137:826-832
Jones RK. Increased intracranial pressure following radical neck surgery. Arch Surg 1951;63:599-603
Schwizer 0,Leak GH. A study of spinal fluid pressures in operations requiring removal of both internal jugular veins.
Intracranial hypertension following neck dissection. J Laryngol Otol 1986;
ChaiPJ, SkaryakLA,Ungerleider RM,etal. Jugular ligation does not increase intracranial pressure but does increase bi- hemispheric cerebral blood flow and metabolism. Crit Care Med 1995;23:1864-1871
CCSVI is most-likely a chronic, lifelong accumulation of injury to the brain, spine, and optic nerve. That's why Dr. Zamboni followed those kids in Sardinia and found that 90% had MS 20 years later.
Dr. Zamboni’s personal journey leading to the rediscovery of the vascular link to MS, and his subsequent elaboration of this connection, began more than twenty years ago, when he was a vascular surgeon in Sardinia, Italy. Sardinia is geographically isolated and, for reasons largely unknown, contains very high rates of MS. Studying children in Sardinia, Dr. Zamboni discovered that a great number of them had malformations in their jugular veins. This work lead to one of his earliest professional publications, “The so-called primary venous aneurysms,” published in Phlebology, in 1990.