This might be relevant...
Ann Neurol. 2011 Jan;69(1):90-9. doi: 10.1002/ana.22228.
No evidence of chronic cerebrospinal venous insufficiency at multiple sclerosis onset.
Baracchini C, Perini P, Calabrese M, Causin F, Rinaldi F, Gallo P.
SourceFirst Neurology Clinic, University Hospital, Padova, Italy. firstname.lastname@example.org
OBJECTIVE: An impaired cerebrospinal venous drainage, defined as chronic cerebrospinal venous insufficiency (CCSVI), has been recently hypothesized to be the possible cause of multiple sclerosis (MS). We investigated this hypothesis by studying the occurrence of CCSVI in clinically isolated syndromes (CISs) suggestive of MS.
METHODS: Fifty consecutive patients presenting with a CIS and evidence of dissemination in space of the inflammatory lesions (ie, possible MS [pMS]) underwent a detailed diagnostic workup, including extracranial and transcranial venous echo-color Doppler sonography (ECDS-TCDS). Those with CCSVI underwent selective venography. Fifty healthy subjects (HCs) age-matched and gender-matched with pMS patients (HC1); 60 patients with transient global amnesia (TGA); and 60 healthy subjects age-matched and gender-matched with TGA patients (HC2) constituted the control groups and underwent ECDS-TCDS.
RESULTS: Mean age of pMS patients was 33.0 ± 8.5 years (range, 14-50); 35 (70%) were female (female:male ratio, 2.3). TCDS was normal in all pMS patients. One or more abnormal ECDS findings were observed in 26 of 50 (52.0%) pMS patients, in 35 of 110 (31·8%) HCs (HC1+HC2), and in 41 of 60 (68.3%) TGA patients.
Eight (16%) pMS patients fulfilled the diagnosis of CCSVI. Selective phlebography performed in 7 of these patients (1 denied consent) did not show venous anomalies.
INTERPRETATION: Our findings do not support a cause-effect relationship between CCSVI and pMS. Further studies are warranted to clarify whether CCSVI is associated with later disease stages and characterizes the progressive forms of MS.
People with transient global amnesia have either one or both jugular valves dysfunctionally allowing blood flow to go both ways. After CCSVI treatment, this is often the case for us as well.
So we might show criteria of reflux on the exam but it's no longer reflux caused by CCSVI which would mean it's not in need of further treatment. More research would clarify the value of the Zamboni protocol ultrasound used after the CCSVI procedure.
I would pay more attention to symptom improvement and any worsening and it sounds like you are doing well in that regard!