Back to my original question, I am wondering how other autoimmune diseases could be affected by the discovery of CCSVI in ms patients. Could venous abnormalities be an underlying risk factor in the development of “autoimmune diseases”?
In the recent 'massive gene study' it was found that a third of the genes common to MS patients are also common to other autoimmune diseases. These genes were said to affect the immune system, but Cheer has shown some research showing that genes can affect both the vascular and immune systems.
I would want to look at lupus and say, what venous abnormalities, where, could have this result? And the same with diabetes mellitus, which looks very similiar to MS genetically. What venous abnormalities, where, how.
Anything in the human body that can go wrong, does go wrong. Sometimes very very rarely, sometimes more commonly. So what can go wrong with the endothelium or the vasculature?
Autoimmune may have become a lumping-together category of illnesses that are not well understood. Absolutely the immune system can go haywire too. But it can also be effectively cleaning up after a different aspect of the body has gone haywire.
As for the original question, it is just too early to know. CCSVI is only just been defined and preliminarily explored. We've yet to know if the implications are broad to include other illnesses or narrow to include MS or more narrow still to include only a subset of MS.