se1956 wrote:Venous leg diseases are often congenital.
First symptoms / problems start in many cases in the age of 20-30.
MS has the same “timing”. This is a strong argument pro CCSVI as a major risk factor for triggering MS but clearly no proof.
EJC wrote:This my question though. Maybe all these "congential" venous diseases aren't congenital, maybe they are being casued by muscular or nerve related problems. It's just no one has ever had casue to investigate as we've all assumed it was congenital.
EJC wrote:My partner underwent treatment for CCSVI in December 2010, like many other patients showed very promising real world physical results most of which gradually faded away again. We have decided to undertake an alternative form of treatment which I am documenting on this forum.
Why? How can a procedure produce such great results for them only to slip away again? Well maybe, just maybe we're treating another symptom.
Cece wrote:Has she had any imaging tests done since the improvements slipped away? Doppler or MRV? The first way to answer that question would be to see if the veins are no longer open.
cheerleader wrote:supposition is fun, but research is best.
http://ms.about.com/b/2011/10/25/a-ccsv ... ink-so.htm
the chicken and egg debate is being elucidated by Dr. B.B. Lee, Dr. Zamboni, Dr. Fox and many others.
truncular venous malformations, such as the ones found upon autopsy, are congenital.
Prevalence of deep venous anomalies in congenital vascular malformations of venous predominance☆☆☆
Sandra Eifert, MD, J.Leonel Villavicencio, MD, Tzu-Cheg Kao, PhD, Bettina M. Taute, MD, Norman M. Rich, MD
Received 19 May 1998; accepted 24 June 1999.
Purpose : The overall incidence of congenital vascular malformations in the general population is 1.5%. Approximately two thirds of them are malformations of venous predominance. Abnormalities of the deep venous trunks have been observed in association with large superficial compensatory varices in these type of malformations. Knowledge of the integrity of the deep venous system is important in their management because excision of the enlarged superficial veins may be deleterious if there is aplasia or hypoplasia of the deep venous trunks. The objective was to investigate the prevalence and nature of deep venous anomalies that occur in patients with congenital vascular malformations of venous predominance both in our series and in the series from the medical literature. Methods : From the last 35 years of medical literature, we reviewed seven series of congenital vascular malformations that provided pertinent information on the subject of our study. We also reviewed our own series of 392 patients with congenital vascular malformations studied at Children's Hospital of Mexico City (1963-1983; n = 223 children) and at Walter Reed Army and National Naval Medical Centers (1984-1998; n = 169 children). Of 392 patients, 257 (65.5%) had malformations of venous predominance; these were the subject of our analysis. Prevalence of the following deep venous anomalies was recorded: phlebectasia, aplasia or hypoplasia of venous trunks, aneurysms, and avalvulia. Diagnosis was made by one or more of the following methods: Doppler scanning, duplex scanning, plethysmography, computerized tomography, magnetic resonance imaging, and angiography. Results : At least one anomaly of the deep venous system was present in 47% of the congenital vascular malformations of venous predominance reviewed. Phlebectasia was recorded in 36% of the cases, and aplasia or hypoplasia of deep venous trunks was observed in 8% of the cases. Venous aneurysms also were present in 8% of the cases; avalvulia was recorded in 7% of the cases. Conclusion : Anomalies of the deep venous system occur in almost one half of congenital vascular malformations of venous predominance. The most common is the relatively innocuous phlebectasias that occur in over one third of cases. Aplasia/hypoplasia, venous aneurysms, and avalvulia were less frequent, each less than 10%; but failure to detect the latter three anomalies may lead to serious therapeutic errors. (J Vasc Surg 2000;31:462-71.)
Our stubborn veins seem to like to recreate the condition of CCSVI all on their own, through elastic recoil, returning to the positions that they are used to.
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