Some authors have reported the occurrence of intra-cerebral hemorrhages that have been caused by venous diseases. These are usually located in the white matter at the border zone between deep and superficial venous systems where collaterals are poor. Venous intra-cerebral hemorrhages are associated with impaired venous hemodynamics, as in the case of cerebral venous thrombosis, compression of the superior cava vein or right cardiac failure . Moreover, the occurrence of petechial hemorrhages during cerebral venous thrombosis is a frequent finding on computed tomography (CT) or magnetic resonance imaging (MRI) scans [4,5].
This is from an article on a older gentleman who had jugular insufficiency of the 'Doepp' kind, by which I mean that instead of having too-thick immobile jugular valves as seen in CCSVI, he had flopsy useless jugular valves that let flow through from the brain to the heart and also let it reflow back from the heart to the brain. After CCSVI treatment, we may acquire this other type of jugular insufficiency, of which there is some concern, although I see it as a lesser-of-two-evils (better to let the flow out, even if under specific conditions it flows back, then to keep the flow in).
This gentleman had a cerebral hemorrhage during sex, probably due to reflow from his heart to his brain due to this jugular insufficiency. I posted this in Cheer's why we need valves thread.
Ok, with all of that out of the way, this quote was interesting! Intracerebral hemorrhages tend to occur in white matter at the border zone between deep and superficial venous systems where the collaterals are poor. Are our white matter lesions in such areas?