Cece wrote:the hypertension at least as discussed in this article was from the compression (outflow obstruction) of the IJVs
compression of the IJVs led to increased pressure in the cerebral capillaries (hypertension of the cerebral capillaries?)
no word yet on if antihypertensives could help the situation
logically I would think it's a possibility!!
But then we run back into that old question. How does CCSVI angioplasty sometimes cause such immediate and noticeable improvements? I don't see release of cerebral capillary hypertension on it's own having such a dramatic, instant effect. Some sort of major brain function has to be improved almost instantly with the release of the pressure. Doesn't it?
I wonder how much they compressed the jugulars and if it is similar to 'compression' in ms ccsvi. Also, if obstruction of cerebral veins leads to their death and resorption, then how would angioplasty undo their demise within minutes? If it only affects veins before they die but while they are sickly, instant noticeable improvements aren't likely either. Angioplasty has to do something bigger. Doesn't it?
Are there studies going on trying to figure out *why* and not *if* angioplasty works?
Do anti-hypertensives dilate stenosed areas too?
So many questions...