HERE is a paper on what the problem is regarding how the jugular stenosis is seen
Quote:
Venous outflow of the brain after bilateral complete jugular ligation.
Ensari S, Kaptanoğlu E, Tun K, Gün T, Beşkonakli E, Celikkanat S, Dere H, Cekirge S.
Ankara Numune Education and Research Hospital, Department of Otorhinolaryngology, Ankara, Turkey.
A case of a patient with bilateral internal, external, posterior external and anterior jugular vein ligations and excisions performed in the neck due to a larynx tumor is presented. Radical neck dissection is a standard otorhinolaryngological procedure in the management of head and neck cancer patients with bilateral lymph node metastasis to the neck. Sacrifice of both internal and external jugular veins bilaterally has been recognized as a dangerous approach leading to intracranial hypertension with subsequent neurological sequela and death. In this report, we aimed to demonstrate how venous outflow from the brain diverts after jugular venous system obliteration. After bilateral jugular vein ligations, digital subtraction angiography (DSA) showed that the venous drainage route of the brain had been diverted from the jugular veins to the vertebral venous plexus.
This is pretty well commonly thought that the jugs are not necessary the vertebrals can just take up the slack no problem, but once again these people test it out mere days after they do their work on the person. This one had cancer, did she even live 10 more years? If she did and she had lesions in her brain after time was it attributed to MS or some post cancer thing that just happened? the assumption that it is fine because it looks OK the week after surgery may be very short sighted. The abstracts that follow affirm the idea that an overloaded venous system causes damage contrary to the idea offered here that it works just fine, and in support of CHeers paper that shows taking out the jugs may not be OK
My own stenosis showed significant pressure differences and relief of presssure after the stents were in.
HERE is a paper on venous hypertension causing myelopathy in the cervical cord
Quote:
STUDY DESIGN: Case report. OBJECTIVE: We describe a patient who developed a myelopathy associated with a noncompressive herniated cervical intervertebral disc at the same level. We provide clinical and radiological evidence that reveals that even though the disc herniation did not compress the spinal cord, it diminished venous blood flow out of the spinal cord, possibly resulting in a venous hypertensive myelopathy (VHM). SETTING: Baltimore, MD, USA. CLINICAL PRESENTATION: A 29-year-old woman developed a cervical radiculopathy, followed by a slowly progressive cervical myelopathy associated with a herniated C5-C6 disc. Magnetic resonance imaging showed a noncompressive disc herniation, a swollen spinal cord with increased T2 signal most prominent at the site of the herniated disc, extending several levels above and below the disc. The patient was diagnosed with acute transverse myelitis (ATM) and was started on IV steroids. However, unlike most cases of transverse myelitis, spinal fluid analysis was noninflammatory. In contrast, several features suggested that the patient instead had VHM. We suggest that the disc herniation resulted in impaired drainage of blood from the spinal cord through compression of the venous plexus near the intervertebral foramen. INTERVENTION: Although the patient did not recover function following high-dose steroid administration, she recovered completely following C5-C6 discectomy and fusion. CONCLUSION: To our knowledge, this is the first report of likely VHM in the absence of a spinal arteriovenous malformation. We suggest that some patients diagnosed with ATM in the setting of extrinsic spinal column abnormalities may actually have a noninflammatory myelopathy associated with impaired spinal venous drainage.
ANd a stunner
HEREQuote:
Diagnosis of venous circulation disorders in the cervical portion of the spine and spinal cord using selective phlebography]
[Article in Russian]
Ogleznev KO, Tsuladze II.
Selective phlebography of the major branches of the vena cava superior, supplemented, if necessary, by selective catheterization of the spinal vein, was employed to study the disorders in the cervical section of the spine and cord. Twenty-five patients with myelopathies of obscure origin were examined. The main symptoms were stenoses and atresias of the orifices of the internal jugular veins and compression and stenoses of the brachiocephalic trunks. The possibility of a reverse venous blood stream from the nonspinal veins to the epidural ones and backward was confirmed. Therefore, a disorder of the venous outflow from the spinal channel, on the one hand, and intensification of the venous blood inflow, on the other, may develop under certain conditions, resulting in intrachannel hypertension. Eight patients with pathologic changes in the veins (decompressions and angiolysis) were operated on.
These people had myelopathies caused by venous backflow............ that were fixed after surgery to relieve the atresia (blockage from being formed wierdly) or stenosis (another kind of blockage).
anyone hanging out on this forum knows this is exactly what we are talking about here. 1993. Not specifically MS but "myelopathies of unknown origin"
HEREQuote:
Spinal cord oedema due to venous stasis.
F C Henderson
H A Crockard
J M Stevens
Department of Surgical Neurology, National Hospital for Neurology and Neurosurgery, London, UK.
Venous hypertension and stagnant hypoxia in the human spinal cord are poorly understood. We report a case in which a partial Brown-Séquard syndrome resulted from obstruction of venous drainage on one side of the spinal cord. Neurological deterioration and eventual recovery paralleled the formation and clearance of oedema. The clinical and MRI findings support the contention that spinal cord injury arising as a result of obstruction to venous outflow occurs primarily in the white matter, spreading secondarily to the grey matter, and observations that obstruction of venous flow in the high cervical spinal cord results in changes in the lower cervical cord. MedlineID:
PMID: 8492903
Primarily in the white matter----secondarily to the grey matter.....
Uh, BINGO!!
is this fun or what?
marie
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forgive me Cheer but I would like to pick the paper apart a bit...
It looks like the pieces of the puzzle are starting to fit together or maybe I'm just looking at it with CCSVI colored glasses. 
So I'll take that monkey off your back so you can concentrate on the good stuff that's coming and get yourself over that finish line. I'll leave it to someone else to say it will all work out, you'll get to Stanford and you'll move ahead from there.