sbr487 wrote:Frankly news like this really makes my day.
Dr. Z's research should offshoot into multiple areas. This will ensure:
1) Actual research data is available for everyone (including sceptics)
2) Vascular diseases as underlying issue for lot of diseases will grow into a new discipline of its own
3) It will be difficult for anyone to stop the interest in this area
Great going ...
Johnson wrote:Wow. That is cool.
I found this interesting too - changes in normal appearing white matter in multiple sclerosis are characteristic of neuroprotective mechanisms against hypoxic insult. I have been supposing for a while that the fibrin/scars/lesions are a protective mechanism more than a "normal" scarring - a protection from further injury, rather than a reaction to an injury. I am finding it hard to express the nuance right now...
To shorten urls, try http://www.tinyurl.com. For instance, I did a search for the abstract you outlined, and took it to tiny url for this - http://tinyurl.com/mark-abstract.
Clear as mud.
CureIous wrote:"Angiogenesis" is mentioned + stiffened blood vessel linings. Wonder if that would extend to other veins like in the neck for instance, and at some point, that accounts for loss of flexibility, and inability (all structural considerations aside like bone pushing into it for example) of the vessel to "bounce back", and at that point, the stenosis stays that way, with minimal amount of re-inflation to it's ordinary diameter, almost like the difference between a flexible hose that collapses, but inflates when water goes through it, and a solid copper pipe with a dent in it that doesn't readily assume it's normal shape without angioplasty.
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