I have somewhat noticed a theme of CCSVI supporters being the types who believe in drug company conspiracies, and even that the NMSS is part of a conspiracy to put the brakes on potential MS cures.
On another forum I visit sometimes that I would call kook fringe alternative there's a woman who's talking about doing blood letting to expedite removal of Iron from her brain.
Lyon wrote:Things change and maybe the natural culmination of events is that every special interest site is doomed in the end to be nothing more than a social gathering place?
I still intend to follow CCSVI to the point that it proves itself to MY standards one way or the other but obviously discussing it at thisisms isn't an essential part of following the research.
I don't think the message has changed from the beginning, in that what had been the best part of thisisms was that it was a good place for two sided discussion has turned into an "agree or get the hell out" situation.
Obviously when only one side of an argument is presented it chases off everyone with an open mind and what is left is a big "what she said!" butt patting session, like what thisisms and the CCSVI forums has become.
The overlap of primary CVD and the C282Y mutation consistently increases the risk of developing venous leg ulceration. These data, which have been confirmed in other clinical settings, suggest new strategies for preventing and treating primary CVD.
In the present study we have investigated whether HFE gene polymorphism may play a role in the disease process of Croatian and Slovenian MS patients and their potential genetic susceptibility to MS. We genotyped 314 MS patients and 400 healthy controls for the C282Y and H63D mutations by polymerase chain reaction/restriction fragment length polymorphism (PCR-RFLP) analysis. Our results showed no significant differences in the distribution of the two mutations between MS patients and controls, suggesting that HFE polymorphisms do not contribute to the susceptibility to MS. Also, there was no significant correlation between HFE polymorphism and the disease progression index. However, we observed that MS patients carrying the mutant C282Y allele exhibited earlier onset of disease symptom relative to other genotypes, but it warrants further study in a larger series of MS patients.
I just wish we could get this forum back to informative posts where we have intelligent discussions about CCSVI,
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