Cece wrote:I thought this might fit here. It's from Dake's presentation at the hamilton conference:
"Lesion etiology is non-specific (congenital/hereditary, osseous impingement, arterial compression, post-inflammatory, arachnoid granulation, etc., alone or in combination)"
This is in reference to the lesions or occlusions in the veins. It sounds like congenital is only one possible source of an occlusion/lesion. Osseous impingement sounds like build-up within the vein. Arterial compression has the carotid artery as the villain. Post-inflammatory...although I'm not familiar with what causes veins to be inflamed. Arachnoid granulation...I do not know what this means.
CCSVI has been classified as a congenital venous malformation, but there would seem to be instances where that is true and other instances where it is acquired.
ozarkcanoer wrote:I find it very interesting about all the different kinds of venous problems that have been encountered by doctors when examining CCSVI patients.
astro wrote:ozarkcanoer wrote:I find it very interesting about all the different kinds of venous problems that have been encountered by doctors when examining CCSVI patients.
Would all of these necessarily show up as reflux on an ultrasound, or would some only be detectable by MRV or venogram?
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