I invite you to look at this wiki page. The page seems to be kept up to date.
http://en.wikipedia.org/wiki/Chronic_ce ... ufficiency
quote: Consequences of Proposed CCSVI syndrome include intracranial hypoxia, delayed perfusion, reduced drainage or catabolites, Increased transmural pressure,  and iron deposits around the cerebral veins.   Multiple sclerosis Has Been Proposed As The main outcome or CCSVI.
Hypoxia is mentioned but strangely enough there is absolutely no mention of low glucose as a possible cause or even a link to the glucose condition.
Therefore, is the current debate on ccsvi as the cause of MS a false debate? Is the low glucose condition consciously kept outside the debate, at least the debate that we
see, while here is precisely the real issue?
This recent link points to a message from a New York neurologist who seems to believe in a glucose link: http://www.ccsvi.mx/fatigue-ms-ccsvi
quote For example, fluctuations in the supply or metabolism of glucose, the primary fuel for the brain, may have some impact on declines ..
This doctor argues: " .. have the neurologists considered the physics of fluid dynamics on the complex bio-chemical interactions that they discuss in their research papers? If so, what was the result? If not, why not?
.. neurologists .. have .. held the field for the last 25 years and it is conceivable that their current scientific background alone is not sufficiently complete to come up with a complete solution."
http://liberationtreatmentccsvi.com/201 ... liberated/
A Google search on hypoglycaemia and MS delivered this fascinating link: Insulin counter-regulation in multiple sclerosis
Clearly there must be massive uploading of this document as Google pushes it forward.
I recognise a number of things in it for myself.
I will look into it further but your views would be most welcome.