I'd refer you to the research of Dr. Philip B. James, University of Dundee Medical School--who began looking at the similarities in decompression sickness during deep dives and neurological illness, specifically MS. He noted spinal degeneration in commercial divers, and suggests HBOT treatments.
http://www.thelancet.com/journals/lance ... 40-6736(88)91167-1/fulltext
He's published much on the subject for the past 30 years.
Quote:
Studies of bubbles formed on decompression in diving have demonstrated the importance of pulmonary filtration in the protection of the nervous system and that filtration is size dependant, as small bubbles may escape entrapment. Fluid and even small solid emboli, arresting in or passing through the cerebral circulation, do not cause infarction, but disturb the blood-brain barrier inducing what has been termed the 'perivenous syndrome'. The nutrition of areas of the white matter of both the cerebral medulla and the spinal cord depends on long draining veins which have been shown to have surrounding capillary free zones. Because of the high oxygen extraction in the microcirculation of the gray matter of the central nervous system, the venous blood has low oxygen content. When this is reduced further by embolic events, tissue oxygenation may fall to critically low levels, leading to blood-brain barrier dysfunction, inflammation, demyelination and eventually, axonal damage.
http://www.ncbi.nlm.nih.gov/pubmed/17439700here is more of his published research--
http://www.ncbi.nlm.nih.gov/pubmed?term ... d=17439700cheer
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Husband dx RRMS 3/07
dx dual jugular vein stenosis (CCSVI) 4/09
dual stents placed 5/09
CCSVI in MS