Scott1 wrote:Hi Anonymoose,
Sorry I'm stubborn. I'm not trying to be. I really don't know enough about IDO to express an opinion. The man in the video said we could discuss the associations for months. So he may not be sure either.
He assumes the EAE mouse model is a model of MS in humans. Not everyone agrees with that but it is a valid assumption if you adopt the autoimmune approach. I'm not sure about it being valid.
I took Valtrex and quickly felt better. I didn't experience the problems (whatever they are I haven't seen described) that others have.
I don't think you measure EBV load. It's more IgG and IgM measures that tell you if you have an infection and whether it is active.
Thanks for answering my questions. I thought valtrex did nothing for you on it's own and only rendered improvement when you took it with avonex. Maybe I am remembering wrong or your evaluation changed with hindsight?
Now, you can't just toss the valtrex ido thing out and tell me to look elsewhere just because you aren't familiar with ido (neither am I btw). It's against the rules, it is! And I think the ambiguity in the video was regarding why hsct renders improvement... not if ipo changes affect disease.
But now that I am distracted with your uric acid obsession, why would I have a relapse because I went from an anti purine to a purine med? Wouldn't the additional uric acid/superoxide eating make things better? Also, how long did it take between you starting valtrex and developing gout? Do you think by reducing ebv you reduced your use of uric acid leading to a reduced need for purines along with symptom reduction?