hey bob so there really is no reason that the drugs are singled out mostly for rr ms other than the fact that people with rr have natural remissions that they can claim the drugs caused.
you talk about plasticity and the ability to heal so someone with bad ms means they just can't heal anymore, there is just to much axonal damage.
so in my 15 years there
has always been inflammation just a slow mild attack that has eventualy left me where i am.
all i'm getting at is how can these drugs not do the same for someone with spms as rr ms. they would still help stop whats going on and leave the person with what permenant damage they have but the progression would stop.
i just don't understand how there is a reason why someone would not get a drug because they are not active
were all active weather were at the start or the end, it's all the same.
here is the inclusion criteria for the campath phase3 trial
Diagnosis of MS and cranial MRI scan demonstrating white matter lesions attributable to MS within 5 years of screening
Onset of MS symptoms within 5 years of screening
EDSS score 0.0 to 3.0 WHY WHY WHY ?????
http://www.genzymeoncology.com/onc/clin ... MMS323.asp
≥2 MS attacks occurring in the 24 months prior to screening, with ≥1 attack in the 12 months prior to screening
Had ms for over 19 years now.