Conclusions: A reduction of BE levels was present in patients with clinically inactive MS. Treatment with IFN-b seems to induce an increase of this opioid in PBMCs of MS patients. The increase of BE concentration during a clinical relapse may represent a possible control mechanism aimed at counterbalancing the inflammatory phase of the disease.
Have either of you tried LDN? this all sounds similar to its method of action.Wonderfulworld wrote:Wish my system made more beta endorphins because when they do hit, I can do nearly everything I want to. But just wish I could avoid the relapse that comes with it nearly every time.
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