ljelome wrote:
... Should those two balance each other?
And how do your theory of excess insulin fit with other people using steroid n managed to reduce their symptoms? (me for example, i always regain my strength after having IV steroid everytime i have relapse and also it helped me with my optic neuritis). Cut i find that steroid only effective for me if i used it immediately after i noticed i had an attack. (maybe because steroid do help overcome the inflammation that's happening).
Insulin and glucose work in tandem, like a seesaw – glucose in the bloodstream causes insulin to go up (but the body always overcompensates for the amount of glucose). The excess insulin that remains after the glucose amount is normalize starts the MS cascade, in my opinion.
Here is how I explain your experience with steroids: Too much insulin was damaging the small blood vessels in your eye (causing optic neuritis); the IV steroid created more glucose in your body, which temporarily "busied" the insulin and reduced the insulin in your bloodstream (thereby, temporarily stopping insulin's damage).
But, your body detects the additional glucose created by the steroid; and your body produces more insulin (again overcompensating and more insulin will produce more symptoms after a while).
Scientists have documented an increase in diabetes in those people who use steroids many times or over a long period of time. I think this is also because of this back and forth battle between glucose and insulin.
This is the possibility I see.
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