speaking of eyes...
Iron homeostasis and eye diseasehttp://www.ncbi.nlm.nih.gov/pmc/articles/PMC2718721/
"Iron is necessary for life, but excess iron can be toxic to tissues. Iron is thought to damage tissues primarily by generating oxygen free radicals through the Fenton reaction. We present an overview of the evidence supporting iron's potential contribution to a broad range of eye disease using an anatomical approach. Firstly, iron can be visualized in the cornea as iron lines in the normal aging cornea as well as in diseases like keratoconus and pterygium. In the lens, we present the evidence for the role of oxidative damage in cataractogenesis. Also, we review the evidence that iron may play a role in the pathogenesis of the retinal disease age-related macular degeneration. Although currently there is no direct link between excess iron and development of optic neuropathies, ferrous iron's ability to form highly reactive oxygen species may play a role in optic nerve pathology. Lastly, we discuss recent advances in prevention and therapeutics for eye disease with antioxidants and iron chelators. ... Although further research is necessary, advances in understanding the role of oxidative damage in optic neuritis may lead to novel uses for antioxidant therapy and potentially for discovering a link between iron-related production of ROS and optic neuritis....."
with a bit of searching we could probably draw a line max three to four studies long between high iron, low zinc, and optic neuritis.