Sharon wrote:Hey Cheer -
I will be the trial for thisisms. I have been taking LDN since May of 2008. I have never taken any other drug for my MS. I have suffered from vascular problems for years (varicose veins for which I have had operations) I am hesitant to report anything at this time, except to say that I am not any worse than I was six months ago (yea!), and a friend of mine has commented that my spider veins are not as prominent. She was wondering what doctor I was seeing for vein treatment. I was happy to tell her "no doctor" - just supplements, diet and LDN. The LDN seems to be working for me - I am noticing more benefit from my exercise program, and my energy level has increased.
varicose vein sections showed increase in the diameter of the lumen, hypertrophy of the wall and elongation and invagination of the intima. Along these invaginations, endothelial cells were compressed, elongated and thinned out. The cells also showed progressive degeneration and were finally lost into the lumen, leaving only the basal lamina to form the luminal surface. This invited blood components like platelets and red blood cells to stick to the bare intima and to penetrate through the wall
The only thing that's holding me back from doing it again is the amount of preparation and hunger I had to face. Little son takes up most of my energy, not a lot left for sourcing and baking food.
3rd trimester I felt SO awful I've no idea if my blood was flowing well. I was exhausted and sick but perhaps my blood was good
DIM wrote:Cheer if my memory serves me right you have DHEA in husbands regimen correct?
I found that DHEA which declines remarkably after 35-40 years in mens plays critical role on endothelium healing dysfunctions and is one of the reasons it helps so much in cases of cardiovascular problems!
The workshop kicked off by considering one of the most important molecular processes common to a number of cancers and cardiovascular disease, involving the pathway known as the endothelin axis. The endothelium is the thin layer of cells lining every blood vessel of the body from the smallest capillaries to the largest arteries and even the heart itself. This layer separates the blood from the vessel walls and the smooth muscles whose contractions restrict and control blood flow. These muscle contractions are controlled by proteins called endothelins manufactured by the endothelium cells, and if there are too many of them blood flow is restricted too much, leading to hypertension (high blood pressure) and participating in other conditions such as acute coronary syndrome and stroke.
High-altitude pulmonary edema (HAPE) is characterized by excessive pulmonary vasoconstriction and is associated with decreased concentrations of nitric oxide (NO) in the lung Conclusions: Hypoxia markedly impairs vascular endothelial function in the systemic circulation in HAPE-S subjects due to a decreased bioavailability of NO. Impairment of the NO pathway could contribute to the enhanced hypoxic pulmonary vasoconstriction that is central to the pathogenesis of HAPE.
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