Nerve conduction, sugar and fat..
Posted: Mon Nov 14, 2011 1:32 am
MS patients have a bad nerve conduction of signals from the brains to the periphery and vice-versa. The problem of conduction is serial: there is a problem with the conduction in the CerebroSpinal (due to the scleroses) and in addition conduction may degrade because of pre-diabetic conditions. For diabetes patients, the neurological symptoms are similar to ours like numbness of the hands etc. For them, these symptoms show up when the diabetes diagnosis is made but then the process is already underway for many years. For us, these symptoms show up much earlier because we already have an impaired conduction of the signals. Therefore, we are much more vulnerable to diabetes-like nerve degeneration.
Besides demyelination, I believe the degradation is mainly caused by an undernourishment of the cells, including nerve cells with the result that the ion pump is not sufficiently charged to maintain its equilibrium necessary for passing nerve signals and ultimately - if undernourished for a long time - the death of the cell. In this context, I believe that the combination of (thick) saturated fats and high sugar consuption is devastating. I also believe that it is no so much the arteriosclerose or hardening of the veins, but rather that it is the GLUTS (the glucose transporters on the cells at micro-cellular level) that are blocked-off that is the problem. This would explain why the Swank diet works and also why the no-sugar diet (as the old wise neurologist said) works to turn away from the MS disease. I think the GLUT problem really applies to us because we have a problem with the power of the cells. And what we have left, we must keep alive.
I think that MGmin cholesterol that grows on sugar and thick saturated fats and that "seals" the GLUTS of the cells is somewhere somehow part of the picture. As well as Metformin that seems to be able to break down the ultra-bad and super-sticky MGmin cholesterol. I have seen many working mechanisms for Metformin suggested including stabilising the blood sugar level/glucose level and working on the liver to produce lower peaks in the blood sugar. I think the medical sector has to leave behind another dogma here. And accept that it is the cells (at micro-cellular level) that open up and absorb the glucose better due to the medication. Thereby it will look as if the blood sugar is regulated down but probably that is conceptually wrong.
This pre-diabetic condition (which is causing the progressive phase of MS) is more likely to happen at mid-age because of the skin (lower Vitamin D production), veins (hardening) and intestinal flora unbalances (Western diet and lack of Vitamin D). I think this is a wonderful article http://www.healingmatters.com/diabetes.htm describing the important role of the GLUTs.
Besides demyelination, I believe the degradation is mainly caused by an undernourishment of the cells, including nerve cells with the result that the ion pump is not sufficiently charged to maintain its equilibrium necessary for passing nerve signals and ultimately - if undernourished for a long time - the death of the cell. In this context, I believe that the combination of (thick) saturated fats and high sugar consuption is devastating. I also believe that it is no so much the arteriosclerose or hardening of the veins, but rather that it is the GLUTS (the glucose transporters on the cells at micro-cellular level) that are blocked-off that is the problem. This would explain why the Swank diet works and also why the no-sugar diet (as the old wise neurologist said) works to turn away from the MS disease. I think the GLUT problem really applies to us because we have a problem with the power of the cells. And what we have left, we must keep alive.
I think that MGmin cholesterol that grows on sugar and thick saturated fats and that "seals" the GLUTS of the cells is somewhere somehow part of the picture. As well as Metformin that seems to be able to break down the ultra-bad and super-sticky MGmin cholesterol. I have seen many working mechanisms for Metformin suggested including stabilising the blood sugar level/glucose level and working on the liver to produce lower peaks in the blood sugar. I think the medical sector has to leave behind another dogma here. And accept that it is the cells (at micro-cellular level) that open up and absorb the glucose better due to the medication. Thereby it will look as if the blood sugar is regulated down but probably that is conceptually wrong.
This pre-diabetic condition (which is causing the progressive phase of MS) is more likely to happen at mid-age because of the skin (lower Vitamin D production), veins (hardening) and intestinal flora unbalances (Western diet and lack of Vitamin D). I think this is a wonderful article http://www.healingmatters.com/diabetes.htm describing the important role of the GLUTs.