This Is MS Multiple Sclerosis Knowledge & Support Community

Nenu wrote:

Andesine wrote:Sally, my MS dx started with Double Vision, light sensitivity, weird sort of pre migraine shapes and light that never progressed further.

Do you mean you never developed the migraine following the shapes? I certainly experienced this myself, back in 2001. Years before my MS dx.

This is an excellent example of what happened in my case...Imagine this in 1 eye, and that's what would come on and then disappear, over approx 30 minutes (as shown in this particular example)

cheerleader wrote:Andesine and Nenu-
You're describing optic migraine...which only affects the vision, there's no headache afterword. I've had them for four years, and get them before my period. I used to get classic migraine with aura as a young adult. They went away in my thirties. Funny thing is, I've had a couple MRIs and all the MS tests before I even knew about this disease, or before my husband was diagnosed. Our joke is that he always "has to do one better" than me! I just have migraine, but he has MS!!!

AC

"Knowledge is Power"

Multiple sclerosis (MS) has a much higher incidence among caucasians that in any other race. Furthermore: females are much more susceptible than males and white females living in colder, wetter areas are much more susceptible than those living in warmer areas. On the other hand, menstruating women have increased copper (Cu) absorption and half-life, so they tend to accumulate more Cu than males. Moreover, rapidly growing girls have an increased demand for zinc (Zn), but their rapidly decreasing production of melatonin results in impaired Zn absorption, which is exacerbated by the high Cu levels. The low Zn levels result in deficient CuZnSuperoxide dismutase (CuZnSOD), which in turn leads to increased levels of superoxide. Menstruating females also often present with low magnesium (Mg) and vitamin B6 levels. Vitamin B6 moderates intracellular nitric oxide (NO) production and extracellular Mg is required for NO release from the cell, so that a deficiency of these nutrients results in increased NO production in the cell and reduced release from the cell. The trapped NO combines with superoxide to form peroxinitrite, an extremely powerful free radical that leads to the myelin damage of MS. Iron (Fe), molybdenum (Mo) and cadmium (Cd) accumulation also increase superoxide production. Which explains MS in males, who tend to accumulate Fe much faster and Cu much less rapidly than females. Since vitamin D is paramount for Mg absorption, the much reduced exposure to sunlight in the higher latitudes may account for the higher incidence in these areas. Moreover, vitamin B2 is a cofactor for xanthine oxidase, and its deficiency exacerbates the low levels of uric acid caused by high Cu levels, resulting in myelin degeneration. Finally Selenium (Se) and vitamin E prevent lipid peroxidation and EPA and DHA upregulate CuZnSOD. Therefore, supplementation with 100 mg MG, 25 mg vit B6, 10 mg vit B2, 15 mg Zn and 400 IU vit D and E, 100 μg Se, 180 mg EPA and 120 mg DHA per day between 14 and 16 years of age may prevent MS.