Smoking injures the endothelium in a way that makes it less likely to put up a healing response to the injury of ballooning?
Nonsmokers are more likely to take care of their health, so nonsmokers are more likely to have gone back for follow-up visits and learned that they restenosed?
ok, you could be onto something, Jugular:
http://radiology.rsna.org/content/231/3/831.fullSmoking 10 or more cigarettes daily is associated with a reduced rate of intermediate-term restenosis after lower-limb endovascular interventions.
Here is the researchers explanation for why smoking might have a beneficial effect, reducing restenosis. This is on lower limb venoplasty with or without stent placement.
But smoking is still terrible for MS, neurologically.Smokers typically have elevated levels of carboxyhemoglobin and higher blood concentrations of carbon monoxide (11,12). Carbon monoxide has potent antiinflammatory and antiproliferative capacity, and it inhibits vascular smooth muscle cell proliferation after vascular injury due to balloon dilation (13–16). Furthermore, cigarette smoke extract induces necrosis in proliferating vascular smooth muscle cells (17). Localized proliferationof vascular smooth muscle cells is a key factor in luminal diameter decrease after endovascular treatment (18). On the basis of these findings, we hypothesized that smoking may protect vessels from restenosis after lower-limb endovascular interventions, presumably through the antiproliferative effect of increased carbon monoxide levels.
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