Mycobacterium avium subspecies paratuberculosis (MAP) vs CPn
Posted: Wed Apr 12, 2017 8:41 pm
Hi all
Have just discovered the Mycobacterium avium subspecies paratuberculosis (MAP) thesis as possibly the major cause of Crohn's disease and ulcerative colitis cases, and of course Red Hill Biopharmaceuticals are also claiming it may be the causative pathogen in MS cases.
I've recently trialled a few patients with MS with the Wheldon protocol which uses abx geared to destroying Chlamydophila pneumonia (CPn) as the presumed most likely cause, with good results so far. These is some overlap in the choice of abx to kill either of these microorganisms, however the Wheldon protocol is the milder protocol of the two and it is also better at killing CPn, rather than MAP. RHB-104 is better at killing MAP than CPn, although antituberculotic drugs work against CPn somewhat.
Given the problems of retinitis, uveitis, macular degeneration and so forth often associated with MS, where CPn has been found in the blood vessels of the eye in sufferers, what is the view on the role of MAP vs CPn as the most likely pathogen in the majority of cases of MS?
Which one is the red herring?
Cheers
Have just discovered the Mycobacterium avium subspecies paratuberculosis (MAP) thesis as possibly the major cause of Crohn's disease and ulcerative colitis cases, and of course Red Hill Biopharmaceuticals are also claiming it may be the causative pathogen in MS cases.
I've recently trialled a few patients with MS with the Wheldon protocol which uses abx geared to destroying Chlamydophila pneumonia (CPn) as the presumed most likely cause, with good results so far. These is some overlap in the choice of abx to kill either of these microorganisms, however the Wheldon protocol is the milder protocol of the two and it is also better at killing CPn, rather than MAP. RHB-104 is better at killing MAP than CPn, although antituberculotic drugs work against CPn somewhat.
Given the problems of retinitis, uveitis, macular degeneration and so forth often associated with MS, where CPn has been found in the blood vessels of the eye in sufferers, what is the view on the role of MAP vs CPn as the most likely pathogen in the majority of cases of MS?
Which one is the red herring?
Cheers