Now I know what happens if we ever miss the point. It gets bolded for us.drsclafani wrote:Perhaps i have not clearly described how important I think the case that i have shown is. In my book it is a smoking gun, a veritable proof that venous obstructions cause symptoms in the absence of prior neurological disease. In essence what has been described is the acute volume overload of the cerebrospinal circuits resulting in neurological symptoms that respond just as as fast as they developed by improvement in venous outflow.
This patient has NO MS, has No neurological disease, No evidence of demyelination. In fact while her ccsvi venous pathology is similar to that of anyone else who has ccsvi, it did not cause symptoms until flow was increased by occluding the ovarian vein and shunting the blood into the cerebrospinal circui and it resolved as soon as angioplasty of the jugulars and stenting of the renal vein was accomplished.
in other words, it is its own symptom complex
I had thought about the ways in which your colleagues might be resistant to some of your ideas demonstrated in this case
* interrogating the dural sinus
* treating a patient without an MS diagnosis
* considering renal stenosis as contributory to ccsvi
* considering IVUS essential to diagnosis and treatment
I recently heard from someone with Lyme disease who could not get her closest CCSVI doctor to test her for CCSVI because she did not have MS, and he is only treating MS patients.
For CCSVI to have its own symptom complex is very significant. Does this case demonstrate such? There are those who dispute that the renal vein stenosis is affecting the cerebrospinal drainage, but I think that has been shown logically and through images of flow going into the ascending lumbars and hemiazygous.
If my CCSVI symptoms were called MS, and my CCSVI has now been treated and those symptoms are gone, what becomes of my MS?
removed by PTA what disease is left? 
