Dear Doctor, thanks for these informations. I understand a little bit better my problems.
I forgot to tell about an other symptom : Very rarely, when I am lying and I am making an effort to stretch a leg, I have a pain, which disappears quickly on the back of my skull, as if the pressure increased and decreased. It is hard to describe exactly.
I will try to get informations about the chiropractors found in my region.
CCSVI and CCVBP
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Kathryn333
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Hi, Drs. Flanagan and Koontz,
I’ve seen Dr. Sondhi twice this week. I know I didn’t absorb everything he said, and I hoped to get a better understanding of his findings before I posted this. He was very busy yesterday, but he said we would go over it next time, and he might even write something for me to post. I told him he he should join us on the website.
Here is my very basic summary from the appointments. In the consultation, he asked about childhood illnesses, surgeries, dental work, if I used sugar substitutes. When he examined my back, he identified six blockages that he felt he could fix. He took x-rays, but doesn’t use the larger x-rays. Dr. Flanagan, you wanted to see the x-rays. I’ll speak to Dr. Sondhi about getting copies to post them when I see him at the end of the week.
He said he could help me. It would take about six months, 20 treatments, and I should be feeling better in 2-3 weeks.
I like Dr. Sondhi very much. I feel so fortunate that he’s treating me. I knew upper cervical specialists were few and far between, but there are only five specialists in Ontario, with two being where I live. How lucky can I be!
The treatment is nothing like any chiropractic adjustment I’ve had before. It’s gentle, subtle, painless, very precise and very specialized.
Thank you again for the interest you’ve both taken and the knowledge you’ve provided on this site. We’ve all learned so much from your postings.
I’ve seen Dr. Sondhi twice this week. I know I didn’t absorb everything he said, and I hoped to get a better understanding of his findings before I posted this. He was very busy yesterday, but he said we would go over it next time, and he might even write something for me to post. I told him he he should join us on the website.
Here is my very basic summary from the appointments. In the consultation, he asked about childhood illnesses, surgeries, dental work, if I used sugar substitutes. When he examined my back, he identified six blockages that he felt he could fix. He took x-rays, but doesn’t use the larger x-rays. Dr. Flanagan, you wanted to see the x-rays. I’ll speak to Dr. Sondhi about getting copies to post them when I see him at the end of the week.
He said he could help me. It would take about six months, 20 treatments, and I should be feeling better in 2-3 weeks.
I like Dr. Sondhi very much. I feel so fortunate that he’s treating me. I knew upper cervical specialists were few and far between, but there are only five specialists in Ontario, with two being where I live. How lucky can I be!
The treatment is nothing like any chiropractic adjustment I’ve had before. It’s gentle, subtle, painless, very precise and very specialized.
Thank you again for the interest you’ve both taken and the knowledge you’ve provided on this site. We’ve all learned so much from your postings.
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blossom
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upright dr.
bump bump!
Hi Dr.'s when you do your adjustment for atlas for instance, what is happening. I am making an assumption here that it is muscle that supports and holds the position of the Skull. If there is misalignment I would surmise that the muscles are things that need to be reset to hold the position of the skull and spine. I am also assuming that the disc material would have a 'memory' and that the click is stretching the disc material to achieve a different position, when the extension happens the muscles would also have to release the tension or extension and the neutral/correct alignment 'could' return. When I have my adjustments the Chiro spends time massaging prior to adjustment and checks movement afterwards.
My active mind assumes that if there is a reason for the muscles to re-spasm or protect a pinched nerve or generally return to the misaligned position because of 'protection mode' that the adjustment would not maintain.
Am I somewhere close with this thinking?
I am also wondering what can happen because of MS related posture changes and general alignment getting worse, and likely effecting blood and CSF flows? Its like we perpetuate the problem.
Regards Nigel.
My active mind assumes that if there is a reason for the muscles to re-spasm or protect a pinched nerve or generally return to the misaligned position because of 'protection mode' that the adjustment would not maintain.
Am I somewhere close with this thinking?
I am also wondering what can happen because of MS related posture changes and general alignment getting worse, and likely effecting blood and CSF flows? Its like we perpetuate the problem.
Regards Nigel.
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uprightdoc
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Hello Blossom, I was wondering about you. Did you get my email?
NZer1, Muscle spasms can be a consequence of misalignment ans much as a cause of it. Alignment is maintained by bones, mucles and connective tissues. Misalignments reoccur due daily stresses and strains as well as inherent structural problems and weaknesses. Many of you have had severe trauma to your spine with permanent damage to the bones, cartilage and connective tissues. Some of you have neurological weakness that predisposes you to misalignments. Lastly, some of you were born with design problems such as scoliosis and kyphosis. I hope tha answers your question.
NZer1, Muscle spasms can be a consequence of misalignment ans much as a cause of it. Alignment is maintained by bones, mucles and connective tissues. Misalignments reoccur due daily stresses and strains as well as inherent structural problems and weaknesses. Many of you have had severe trauma to your spine with permanent damage to the bones, cartilage and connective tissues. Some of you have neurological weakness that predisposes you to misalignments. Lastly, some of you were born with design problems such as scoliosis and kyphosis. I hope tha answers your question.
Thanks Dr. it does. After reading your latest blog post I can see where I fit into your understanding of MS, thanks. I have the view that in general my alignment is historic from leg length, injury and aggravated from time to time by movement, and compounded by posture issues caused by my loss of strength and MS sensory and motor dysfunction.
Great post, thanks again, Nigel
Chronic Ischemia and MS Lesions
uprightdoctor | October 23, 2010 at 8:52 am | Categories: Alzheimer's, Parkinson's, amyotrophic lateral sclerosis, ccsvi, demyelination, ms lesions, multiple sclerosis, primary lateral sclerosis | URL: http://wp.me/p11lYi-AJ
Carotid and Vertebral Arteries
According to Schelling venous back jets into the brain and shear stresses in the cord combined with cerebrospinal fluid reflux are likely causes of demyelination. Other researchers have suggested that chronic edema and normal pressure hydrocephalus can damage myelin simply by stretching it. Zamboni suggests that venous drainage problems lead to iron accumulation, which initiates neurodegenerative processes. There is still another theory that suggests that demyelination may be due to chronic ischemia, that is, decreased blood flow to the brain and cord. For now, I will focus on the blood supply to the brain and ignore the cord.
As depicted it the picture above, blood is delievered to the brain via the anterior and posterior blood supply routes. The anterior blood supply routes are the internal carotid arteries. They are called anterior because they are in the front of the neck. The posterior blood supply routes are the vertebral-basilar arteries in the back of the neck. The carotid arteries travel along either side of the trachea (windpipe) and enter the brain through an opening in the base of the skull called the foramen lacerum where it passes through the cavernous sinus of the drainage system of the brain.
suboccipital cavernous sinus
As shown in the picture above, the vertebral arteries pass through holes in the transverse processes of the cervical spine. They exit the spine at the first cervical vertebra and enter into a memebrane between the base of the skull and the first cervical vertebra. The membrane is called the suboccipital cavernous sinus or atlantooccipital membrane as seen in the picture on the right.
verterbral arteries in the foramen magnum
After traveling through the suboccipital cavernous sinus for a short distance the vertebral arteries turn in and head upward. They next passes through the foramen magnum as shown in the picture on the left. Shortly after passsing through the foramen magnum the vertebal arteries unite to become the basilar artery. Before uniting the vertebral artery gives off two branches. One is called the posterior inferior cerebellar artery because it supplies the lower rear aspect of the cerebellum. The other artery is called the anterior spinal artery.
The two anterior spinal arteries then unite and head back down through the foramen magnum to supply the entire length of the anterior aspect of the spinal cord. Another artery called the posterior spinal artery branches off either the posterior inferior cerebellar arteries or the anterior spinal arteries to become the posterior spinal artery. The posterior spinal artery supplies the posterior aspect of the entire length of the cord. The cord also gets its blood supply from what are called radicular arteries. For now we will overlook the possible role of the anterior and posterior spinal arteries in chronic ischemia of the spinal cord and focus on the vertebral-basilar arteries and ischemia of the brain. In brief, chronic ischemia of the cord may play a role in amyotrophic lateral sclerosis and primary lateral sclerosis.
vertebral-basilar arteries
The vertebral-basilar arteries supply most of the the motherboard of the brain, which is the brainstem and cerebellum. Except for the nose and eyes it also supplies all the cranial nerves, which control the muscles and senses of the head and neck down to the shoulders as well as autonomic function of the internal organs via the vagus cranial nerve. In this regard, the vertebral-basilar arteries also supply the hypothalamus, which is the control center for autonomic vegetative functions in the body, and the thalamus, which is the sensory switchboard or router of the brain.
In additon to the brainstem and cerebellum, the vertebral-basilar arteries supply the inner parts of the temporal, and the inner parts and rear pole of the occipital lobes of the brain. Among other things, the inner temporal lobe is important to memory. The occipital lobe is important for interpreting visual signals which is likewise dependant upon memory.
The areas of the brain at the tail ends of the anterior and posterior blood supply routes are called the watershed areas of the brain. The watershed areas of the brain are the weakest point in the blood supply routes and oftentimes are the ones most affected by decreases in blood flow. Like the brain the cord also has watershed areas which I will leave out of this discussion. One of the watershed areas in the brain are around the periventricular areas where MS lesions are often found. Alzhiemer's and migraine headaches also have a prediliction for periventricular white matter lesions. Many researchers believe that the white matter lesions in MS, migraines and Alzheimer's disease are caused by chronic ishemia.
While the lesions are typically found higher up in the brain above the covering over the cerebellum, many of the symptoms associated with MS come from cranial nerves supplied by the vertebral-basilar arteries. Symptoms such as dizziness and fatigue are mostly likely signs of ischemia. Likewise the lesions seen in certain cases of MS, especially those assoicated with migranous symptoms are most likely caused by chronic ischemia. What's more, the vertebral-basilar arteries are the ones most commonly involved in mini strokes called trainsient ischemic attacks. The design of the skull, spine and circulatory system of the brain predisposes humans to upper cervical strains and subsequent compression and back pressure against the posterior blood supply routes, which are the vertebral-basilar arteries. The outcome is chronic ischemia in the areas of the brain they supply.
In my next post I will discuss upper cervical strains and deformation of neurovascular tunnels. Upper cervical strains and deformation of critical neurvascular tunnels are most likely one of the major causes of chronic ischemia and edema in the brain. They may also play a role in normal pressure hydrocephalus, but that's an entirely different story more related to Parkinson's and Alzheimer's disease.
Great post, thanks again, Nigel
Chronic Ischemia and MS Lesions
uprightdoctor | October 23, 2010 at 8:52 am | Categories: Alzheimer's, Parkinson's, amyotrophic lateral sclerosis, ccsvi, demyelination, ms lesions, multiple sclerosis, primary lateral sclerosis | URL: http://wp.me/p11lYi-AJ
Carotid and Vertebral Arteries
According to Schelling venous back jets into the brain and shear stresses in the cord combined with cerebrospinal fluid reflux are likely causes of demyelination. Other researchers have suggested that chronic edema and normal pressure hydrocephalus can damage myelin simply by stretching it. Zamboni suggests that venous drainage problems lead to iron accumulation, which initiates neurodegenerative processes. There is still another theory that suggests that demyelination may be due to chronic ischemia, that is, decreased blood flow to the brain and cord. For now, I will focus on the blood supply to the brain and ignore the cord.
As depicted it the picture above, blood is delievered to the brain via the anterior and posterior blood supply routes. The anterior blood supply routes are the internal carotid arteries. They are called anterior because they are in the front of the neck. The posterior blood supply routes are the vertebral-basilar arteries in the back of the neck. The carotid arteries travel along either side of the trachea (windpipe) and enter the brain through an opening in the base of the skull called the foramen lacerum where it passes through the cavernous sinus of the drainage system of the brain.
suboccipital cavernous sinus
As shown in the picture above, the vertebral arteries pass through holes in the transverse processes of the cervical spine. They exit the spine at the first cervical vertebra and enter into a memebrane between the base of the skull and the first cervical vertebra. The membrane is called the suboccipital cavernous sinus or atlantooccipital membrane as seen in the picture on the right.
verterbral arteries in the foramen magnum
After traveling through the suboccipital cavernous sinus for a short distance the vertebral arteries turn in and head upward. They next passes through the foramen magnum as shown in the picture on the left. Shortly after passsing through the foramen magnum the vertebal arteries unite to become the basilar artery. Before uniting the vertebral artery gives off two branches. One is called the posterior inferior cerebellar artery because it supplies the lower rear aspect of the cerebellum. The other artery is called the anterior spinal artery.
The two anterior spinal arteries then unite and head back down through the foramen magnum to supply the entire length of the anterior aspect of the spinal cord. Another artery called the posterior spinal artery branches off either the posterior inferior cerebellar arteries or the anterior spinal arteries to become the posterior spinal artery. The posterior spinal artery supplies the posterior aspect of the entire length of the cord. The cord also gets its blood supply from what are called radicular arteries. For now we will overlook the possible role of the anterior and posterior spinal arteries in chronic ischemia of the spinal cord and focus on the vertebral-basilar arteries and ischemia of the brain. In brief, chronic ischemia of the cord may play a role in amyotrophic lateral sclerosis and primary lateral sclerosis.
vertebral-basilar arteries
The vertebral-basilar arteries supply most of the the motherboard of the brain, which is the brainstem and cerebellum. Except for the nose and eyes it also supplies all the cranial nerves, which control the muscles and senses of the head and neck down to the shoulders as well as autonomic function of the internal organs via the vagus cranial nerve. In this regard, the vertebral-basilar arteries also supply the hypothalamus, which is the control center for autonomic vegetative functions in the body, and the thalamus, which is the sensory switchboard or router of the brain.
In additon to the brainstem and cerebellum, the vertebral-basilar arteries supply the inner parts of the temporal, and the inner parts and rear pole of the occipital lobes of the brain. Among other things, the inner temporal lobe is important to memory. The occipital lobe is important for interpreting visual signals which is likewise dependant upon memory.
The areas of the brain at the tail ends of the anterior and posterior blood supply routes are called the watershed areas of the brain. The watershed areas of the brain are the weakest point in the blood supply routes and oftentimes are the ones most affected by decreases in blood flow. Like the brain the cord also has watershed areas which I will leave out of this discussion. One of the watershed areas in the brain are around the periventricular areas where MS lesions are often found. Alzhiemer's and migraine headaches also have a prediliction for periventricular white matter lesions. Many researchers believe that the white matter lesions in MS, migraines and Alzheimer's disease are caused by chronic ishemia.
While the lesions are typically found higher up in the brain above the covering over the cerebellum, many of the symptoms associated with MS come from cranial nerves supplied by the vertebral-basilar arteries. Symptoms such as dizziness and fatigue are mostly likely signs of ischemia. Likewise the lesions seen in certain cases of MS, especially those assoicated with migranous symptoms are most likely caused by chronic ischemia. What's more, the vertebral-basilar arteries are the ones most commonly involved in mini strokes called trainsient ischemic attacks. The design of the skull, spine and circulatory system of the brain predisposes humans to upper cervical strains and subsequent compression and back pressure against the posterior blood supply routes, which are the vertebral-basilar arteries. The outcome is chronic ischemia in the areas of the brain they supply.
In my next post I will discuss upper cervical strains and deformation of neurovascular tunnels. Upper cervical strains and deformation of critical neurvascular tunnels are most likely one of the major causes of chronic ischemia and edema in the brain. They may also play a role in normal pressure hydrocephalus, but that's an entirely different story more related to Parkinson's and Alzheimer's disease.
Another interest I have is this puzzle is the bodies ability to compensate for damage. The Neuroplasticity work by Norman Doidge (The Brain that Changes Itself ISBN 978-1-921215-82-7) has been of interest to me for some years now. During the time I was seeking a dx I was given a copy by my Muscular Skeletal specialist to help me understand what was happening with the numbness and transitory pains I was having. The damage to brain regions and the severing of nerve tracks in the sensory nerves mostly was something that I could relate to.
I had a gardening injury to my left wrist recently where the nerves to my left thumb were cut. When the cut was stitched the sensory nerves to my left thumb remained damaged. The changes in sensation and the healing process has given my an understanding of the requirements to re-training the sensory system to accommodate the scar and dysfunctions in signal. The sensory signals have not repaired and the messages that I now have to interpret are a challenge.
Touch in particular is in some ways like the signals are back to front, pleasant is now pain and hot and cold are reversed as examples. I still have full motor functions and it is the sensory that are 'confused'.
If this is going on in other scar areas in the CNS it is so easy to understand the issues in my MS.
Places may not be connected but the body corporate is still trying to send and receive communications, and the bloody feels at times like it is a war zone!
I had a gardening injury to my left wrist recently where the nerves to my left thumb were cut. When the cut was stitched the sensory nerves to my left thumb remained damaged. The changes in sensation and the healing process has given my an understanding of the requirements to re-training the sensory system to accommodate the scar and dysfunctions in signal. The sensory signals have not repaired and the messages that I now have to interpret are a challenge.
Touch in particular is in some ways like the signals are back to front, pleasant is now pain and hot and cold are reversed as examples. I still have full motor functions and it is the sensory that are 'confused'.
If this is going on in other scar areas in the CNS it is so easy to understand the issues in my MS.
Places may not be connected but the body corporate is still trying to send and receive communications, and the bloody feels at times like it is a war zone!
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uprightdoc
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Retrospective Case Studies
Hello all,
One of the things I suggest in my book is doing retrospective epidemiological studies of MS patients, which includes a thorough examination of their past history. In this regard I did a preliminary retrospective analysis of fifteen cases that contacted me through this blog. There is still another but I need further information. I copied and pasted then edited your exact words to extract the important history, signs and symptoms. It took several hours and will probably take another couple of hours to clean it up.
Among other things, I hope to discuss your cases with Dr. Haake regarding the role of inherited and acquired disorders and degenerative conditons of the spine in neurodegenerative diseases. Dr. Noam Alperin of the Dept of Radiology of the University of Miami is already on the same page as me as he has been doing upright MRI studies of cranial hydrodynamics. Cases like yours shed further light and should help steer future research in the right direction.
In brief, out of 15 cases: 6 of you had major and typically multiple traumas to the the spine that resulted in spondylosis (degenerative changes in the spine)and misalignments); 5 of you have scoliosis and other abnormal curvature problems such as cervical kyphois (backward curve in the neck) - several of you with curvature problems have lesions in the cord but not in the brain and several with curvature problems also have thoracic outlet syndrome; the remaining 3 cases have migraine variants which are more likely due to chronic ischemia, not venous drainage problems - one migraine case was also associated with a serious car accident many years earlier; 1 case was diagnosed as a Chiari malformation and MS, scoliosis, Chiari, and Alzheimer's run in the family.
This is more than a statistical coincidence. Spondylosis deforms important neurovascular tunnels. Stenosis causes narrowing of the spinal canal and neurovascular tunnels. Scoliosis and abnormal curvatures cause functional stenosis of the spinal canal as they compress blood vessels around the bends in the spine. Lastly, Chiari malformations and tethered cord syndromes, which I will discuss in future posts are far more common than we currently realize. I will be covering them more on my new website. For those of you with migraines or lesions in the brain or cord make sure you read my latest blog.
One of the things I suggest in my book is doing retrospective epidemiological studies of MS patients, which includes a thorough examination of their past history. In this regard I did a preliminary retrospective analysis of fifteen cases that contacted me through this blog. There is still another but I need further information. I copied and pasted then edited your exact words to extract the important history, signs and symptoms. It took several hours and will probably take another couple of hours to clean it up.
Among other things, I hope to discuss your cases with Dr. Haake regarding the role of inherited and acquired disorders and degenerative conditons of the spine in neurodegenerative diseases. Dr. Noam Alperin of the Dept of Radiology of the University of Miami is already on the same page as me as he has been doing upright MRI studies of cranial hydrodynamics. Cases like yours shed further light and should help steer future research in the right direction.
In brief, out of 15 cases: 6 of you had major and typically multiple traumas to the the spine that resulted in spondylosis (degenerative changes in the spine)and misalignments); 5 of you have scoliosis and other abnormal curvature problems such as cervical kyphois (backward curve in the neck) - several of you with curvature problems have lesions in the cord but not in the brain and several with curvature problems also have thoracic outlet syndrome; the remaining 3 cases have migraine variants which are more likely due to chronic ischemia, not venous drainage problems - one migraine case was also associated with a serious car accident many years earlier; 1 case was diagnosed as a Chiari malformation and MS, scoliosis, Chiari, and Alzheimer's run in the family.
This is more than a statistical coincidence. Spondylosis deforms important neurovascular tunnels. Stenosis causes narrowing of the spinal canal and neurovascular tunnels. Scoliosis and abnormal curvatures cause functional stenosis of the spinal canal as they compress blood vessels around the bends in the spine. Lastly, Chiari malformations and tethered cord syndromes, which I will discuss in future posts are far more common than we currently realize. I will be covering them more on my new website. For those of you with migraines or lesions in the brain or cord make sure you read my latest blog.
Hello Doc,
How many times you have come across cases where patients shows migrane as symptoms and has had a history of accident(s)?
A friend of mine complains of migraine regularly and believe it or not, he blames it the accident he had as a kid where his father dropped him on the stairs. I never took it serisouly until I heard about restricted blood, csf flow ... my take was that an injury of that nature would cause far more serious issues than just headaches and most of the issues would be present all the time ...
Also, any comment why people go on to develop different type of symptoms/diseases (like migraines, MS etc.) even though the common cause is anbormal blood or csf flow?
Thank you ...
How many times you have come across cases where patients shows migrane as symptoms and has had a history of accident(s)?
A friend of mine complains of migraine regularly and believe it or not, he blames it the accident he had as a kid where his father dropped him on the stairs. I never took it serisouly until I heard about restricted blood, csf flow ... my take was that an injury of that nature would cause far more serious issues than just headaches and most of the issues would be present all the time ...
Also, any comment why people go on to develop different type of symptoms/diseases (like migraines, MS etc.) even though the common cause is anbormal blood or csf flow?
Thank you ...
A new scientific truth does not triumph by convincing its opponents and making them see the light, but rather because its opponents eventually die and a new generation grows up that is familiar with it
- Max Planck
- Max Planck
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uprightdoc
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There is a significant relationship between trauma and migraines, MS, Parkinson's, Alzheimer's and Chiari malformations.
The signs and symptoms that develop depend on the type of tissue that are affected. Acute severe strokes show instantaneous signs and symptoms. Transient ischemic attacks or mini strokes can be silent and do damage over many years. Aside of fracture and severe tears, damage to bones, connective tissues and cartilage oftentimes breakdown slowy after injuries over the course of many years and take their toll in adulthood. The type of neurodegenerative process is also affected by the circulatory system involved. In some cases it's back pressure against the veins. In others it is due to decreased arterial flow. In still other cases it is more due to decreased CSF flow sometimes known in scientific circles as the "third circulation." It is the most mysterious and critically important. Correct CSF volume is necessary to prevent hydrocephalus and Chiari malformations.
The signs and symptoms that develop depend on the type of tissue that are affected. Acute severe strokes show instantaneous signs and symptoms. Transient ischemic attacks or mini strokes can be silent and do damage over many years. Aside of fracture and severe tears, damage to bones, connective tissues and cartilage oftentimes breakdown slowy after injuries over the course of many years and take their toll in adulthood. The type of neurodegenerative process is also affected by the circulatory system involved. In some cases it's back pressure against the veins. In others it is due to decreased arterial flow. In still other cases it is more due to decreased CSF flow sometimes known in scientific circles as the "third circulation." It is the most mysterious and critically important. Correct CSF volume is necessary to prevent hydrocephalus and Chiari malformations.
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silverbirch
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Hey Doc migraines its weird I had migraines and right jaw clicking following a car accident for many many years ,then went away for a few years came back when I had a tooth extraction ( under sedation) following tooth extraction migraines returned plus lock jaw for 12 months I was left with migraines and a cracking sound comeing from my neck upon turning left /right still got the neck cracking followed by right shoulder pain as if someone punched the top of my right arm shoulder
Then I had my attack DX with MS but prior to that I never attended any one for migraines just recieved immigrain from Dr Im also told immigrain can cause lesions SINCE attack never had another migraine (18 mts ago)
Im off to Dr Heidi London tomorrow 26th I shall report back
TTFN xx
Then I had my attack DX with MS but prior to that I never attended any one for migraines just recieved immigrain from Dr Im also told immigrain can cause lesions SINCE attack never had another migraine (18 mts ago)
Im off to Dr Heidi London tomorrow 26th I shall report back
TTFN xx
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uprightdoc
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silverbirch
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I took these tablets as my friend said like smarties - the label said 1/2 over 12 hours I was taken two at a time I took them prior to a long car 4/5 hour car journey I always got a mirgraine whilst driving hum... sitting for long periods of time hum , turning head left/right hum.. I held the steering wheel tight my neck would crack pain migraineuprightdoc wrote:Thanks for the report Silverbirch. It all makes sense. It's a good thing you are getting your neck taken care of. Unfortunately it's true, several migraine medications cause ischemia and mini strokes. The current theory behind the use of medications for migraines is wrong to begin with.
I even took them prior to meetings this went on for 8 years I took them before they went out for over the counter sale !!!
I should have seen Dr Heidi hundreads of years ago ......
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costumenastional
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uprightdoc
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Hello Costumenational, transluminal angioplasty is not my field but it is just another treatment option for draining the brain. I know it has to be frustrating but before you consider other options I would wait until you see how you do after the kink in your neck in straightened out as best as possible. While you don't have as many problems with degeneration as others and your curves are normal you have a wicked kink in your neck in a critical area. You can only get better having it straightened out. It will take pressure off of arteries, veins and CSF pathways. It will also take pressure off the brainstem, optic nerve and long track motor nerves to your legs.