makes so much sense, to feel bad once veins are blocked - since blood's job is delivering nutrients and oxygen to the cells of the body!
optimal magnesium would deal with vasospasm too, helping loosen them up to let blood flow through.
some contexts in which magnesium helps deal with vasospasm:
The preventive effect of magnesium on coronary spasm in patients with vasospastic angina
http://www.ncbi.nlm.nih.gov/pubmed/11115460
Mg infusion produces nonsite-specific basal coronary dilatation and suppresses Ach-induced coronary spasm in patients with VSA.
The influence of magnesium on visual field and peripheral vasospasm in glaucoma
http://www.ncbi.nlm.nih.gov/pubmed/7715920
"...magnesium improves the peripheral circulation and seems to have a beneficial effect on the visual field in glaucoma patients with vasospasm."
Magnesium infusion for vasospasm prophylaxis after subarachnoid hemorrhage
http://thejns.org/doi/abs/10.3171/jns.2006.105.5.723
Analysis of the results suggests that MgSO4 infusion may have a role in cerebral vasospasm prophylaxis if therapy is initiated within 48 hours of aneurysm rupture.
The Concentration of Magnesium in Erythrocytes in Female Patients with Primary Raynaud's Phenomenon; Fluctuation with the Time of Year
http://ang.sagepub.com/content/45/4/283.short
"The authors conclude that women with PRP have a significantly lower magnesium concentration in erythrocytes during winter than the healthy controls and that this concentration varied with the season of the year in both groups."
a negative study finding, with analysis:
Serum magnesium levels as related to symptomatic vasospasm and outcome following aneurysmal subarachnoid hemorrhage
http://www.springerlink.com/content/746234r2w6814498/
"We identified no relationship between serum magnesium levels and the development of DIND or outcome following aneurysmal SAH.
Forty (31.5%) patients were hypomagnesemic (less than 1.7 mg/dL) during hospitalization, but no difference in outcome (p=0.185) or development of DIND (p=0.785) was found when compared to patients with normal (1.7–2.1 mg/dL) or high (greater than 2.1 mg/dL) magnesium serum levels."
a closer look at the numbers:
"hypo" = <1.7 mg/dL = < 0.6987 mmol/L
"normal" = 1.7–2.1 mg/dL = 0.6987-0.8631 mmol/L
"high" = >2.1 mg/dL = < 0.8631 mmol/L
the 'normal' range, as i've said, is 0.70-1.10.
by the standard set out in the research, 0.91 mmol/L (2.21 mg/dL) is the minimum level to be considered to have optimal status, and anyone 0.90 (2.19) or lower should be supplementing.
anyone have the full text? i'd be interested to see what percent of the total patient group actually had levels above 2.21.
the fact that the controls did not have optimal magnesium status does not automatically require them to have DIND. since magnesium does 300+ jobs in the human body, they could have had any one of 299+ other things operating in a sub-par manner.
this study is misleading, because all sick patients have suboptimal magnesium status. no wonder they found that the level doesn't make a difference.
one other tip - magnesium is quite calming in the emotional sense, as well as the muscular sense
