Had an idle moment and was swatting up on past posts from Dr. F
One that I can align with is misalignment from injury and whiplash.
"With the above racial skull design differences in mind, let's consider a hypothetical example of a European, Asian and African-American traveling together in a car that gets into a serious motor vehicle accident. All of them get whiplash strains and sprains to their cervical spine. They all experience a change of vision and weakness in the legs. Brain scans show small white spots in the brain of the European and long white spots in the cervical cord of the Asian and African-American. In this case the European passenger is diagnosed with MS. The Asian passenger is diagnosed with Optic Spinal Multiple Sclerosis, and the African-American passenger with Devic's disease. In all three cases, it will be blamed on an immunological problem. When in fact, it is simply a different response to the same traumatic cause.
During whiplash the skull and cervical spine are thrust back and forth, along with the brain inside the cranial vault and eye socket. Racial design differences in the skull determine the response to the displacement of the fluids and tissues of the brain and cord inside the cranial vault and eye socket. It may, therefore, explain the difference in incidence of MS in different races. Interestingly, the incidence of MS in Chinese people appears to be rising as more of them drive cars."
"People living in northern climates are exposed to far more winter related whiplash type traumas than people living in southern climates. Motor vehicle accidents and winter sports and activities such as hockey, skiing and snow mobiling significantly increase the risk and the forces involved in trauma.
When it comes to MS and trauma, however, it's not so much the size of the skull that matters as it is how it stacks up on the cervical spine. Moreover, it is also how the brain stacks up over the large hole in the base of the skull, the foramen magnum. Getting back to race and skull designs, brachycephalic Asian and African designs, as well as skulls with short lengths in their base are more balanced on top of the cervical spine. On the other hand, they stack more of the brainstem over the foramen magnum. This predisposes the brainstem and cerebellum to being pushed, pulled or to sink down into the foramen in a Chiari malformation and or pressure conus type condition."
"Chiari malformations used to be considered a childhood problem for the most part and considered to be a herniation of the brainstem into the foramen magnum thus compressing it. More recent research suggests that the definition should be changed to include any decrease in (CSF) volume which causes the brain to sink and come in contact with the bones of the cranial vault. With this in mind, studies now show that trauma can cause Chiari malformations in adults. What's more, Chiari malformations have also been associated with multiple sclerosis and chronic fatigue syndrome."
"The shape of the skull affects the pitch and layout of its base and the venous drainage system of the brain. The length and width of the base of the skull affects the position of the brain within the cranial vault. Herein lies the crux of the potential impact of race on multiple sclerosis due to skull design.
A short length in the base from the front to the back of the cranial vault, predisposes the brain to crowding and a condition called Chiari malformation in which the cerebellum or brainstem gets pushed down into the foramen magnum. Chiari malformations can block both venous blood and cerebrospinal fluid (CSF) pathways causing CCSVI and hydrocephalus type conditions."
"Interestingly, northern Europeans have a much higher incidence of multiple sclerosis than Asians. On the other hand Asians get a variant form of MS called Optic Spinal Multiple Sclerosis. In addition, African-Americans also have a low incidence of multiple sclerosis but likewise, get a particularly aggressive form of what some consider to be a variant of multiple sclerosis, called Devic's disease. Optic Spinal Multiple Sclerosis and Devic's Disease are identical. Both are associated with optic neuritis and transverse myelitis which are discussed separately on this website. The difference may be due to the distribution of weight and, therefore, the center of gravity over the cervical spine. The European design is the least balanced."
From;
http://www.upright-health.com/race.html
And;
"As the brainstem sinks toward the foramen magnum it pulls other parts of the brain down along with it toward the base of the skull. This raises the risk of similar compression of cranial nerves within the openings of the base of the skull, especially the larger openings. The nerves and blood vessels that pass through these openings can thus suffer a similar fate to the brainstem and its circulatory routes in a Chiari malformation; that is, the cranial nerves and blood vessles can get squeezed into the openings in the base of the skull and get compressed. One of the most vulnerable of the cranial nerves in this regard, appears to be the optic nerve."
"the optic nerve is an outgrowth of the hypothalamus of the brain and is wrapped in the meninges of the brain. In fact, the white part of the visible part of the eye, called the sclera, is actually part of the dura mater, or outer coat of the brain. Lastly, cerebrospinal fluid (CSF) flows through perivascular spaces of the optic nerve the same as it does in the perivascular spaces of the brain."
INTERESTING CONNECTION TO THE HYPOTHALAMUS WHICH HAS BEEN NOTED TO BE INVOLVED WITH VASCULAR INSUFFICIENCY AT BNAC.
"The optic nerve fibers terminate in the lateral geniculate body of the thalamus and the superior colliculus of the midbrain."
AGAIN THE THALAMUS WHICH HAS BEE ASSOCIATED WITH VASCULAR INSUFFICIENCY AT BNAC.
"The second type of optic neuropathy is ischemic neuropathy. Ischemic neuropathy occurs when blood flow to the nerve is reduced to the point that it causes irritation and damage to the nerve. There are several key types. Two types in particular are anterior ischemic optic neuritis and posterior ischemic optic neuritis depending on whether the problem originates inside or outside the cranial vault. In either case they have similar consequences, which is loss of or impaired vision."
"In violent head trauma and whiplash injuries, the brain bounces around inside the cranial vault, which can cause bruises that show up on the opposite side of the trauma. Technically, it is called a contrecoup injury. Cerebrospinal fluid also goes through acceleration and deceleration. Lastly, according to Dr. Franz Schelling venous blood from the body, back jets into the brain. All these forces can be destructive to brain tissues.
Furthermore, whiplash injuries generate tension and compression forces that can cause the brain to herniate into structures in the brain and in the skull. In particular, whiplash injuries have been shown to cause Chiari malformations where the brainstem, herniates, that is, gets pulled by tensile forces into the foramen magnum. Likewise, the optic nerve gets tugged by tensile forces that cause it to get pulled back and forth within the optic canal along with the displacement of the brain inside the cranial vault. Thus tensile forces may similarly cause it to become lodged or injured inside the canal similar to a Chiari malformation. Compression of the optic nerve into the optic canal could result in optic neuritis.
Additionally, structural problems such as genetic design issues can lead to undersized spaces and openings in the base of the skull called hypoplasia, which may affect the dimensions of the optic canal. Furthermore, misalignments of the upper cervical spine, exaggerated head tilts and scoliosis of the spine can displace the brain within the cranial vault and increase pressure on the optic nerve and opthalmic artery in the optic canal.
Lastly, the brain is kept floating above the base of the skull and its openings by CSF, which is mostly water, contained in cisterns strategically placed for protection. The chiasmatic cistern is one of the highest cisterns. Its sits beneath the optic nerve and provides protection from the hard bones of the base of the skull.
When CSF volume in the brain decreases, the brain sinks in the cranial vault. Among other things, when the brain sinks in the vault it can compress the brainstem and cranial nerves.
Due to its frequent association with MS and other similar conditions, it appears that one of the most vulnerable cranial nerves is the optic nerve, as well as the opthalmic artery that travels with it through the optic canal. In certain cases, shifts in the position of the brain may be the cause of compressive optic neuritis."
From;
http://www.upright-health.com/optic-neuritis.html
And;
"The cause of demyelination in multiple sclerosis is still believed to be an internal agent, such as an overactive immune system, an inflammatory reaction, or an attack from an external foreign agent, such as a virus or toxin.
However, according to Dr. Franz Schelling, an Austrian physician and an expert on the subject of multiple sclerosis, there has been very little evidence in the way of white blood cells, such as immunocytes, lymphocytic infiltrations or scavenger cells associated with multiple sclerosis to support the immunological or inflammatory theories. This includes sources from environmental triggers, dietary or other causes. Some researchers believe that when immunological and inflammatory evidence is found, it is more likely to be a reaction to the disease process rather than the cause of it. Dr. Schelling is also known for his theory on backjets.
While demyelination is still considered to be the hallmark of multiple sclerosis, recent research shows that contrary to long held beliefs, it may not be the primary problem. As was stated above, it has been shown that the portion of the nerve inside the myelin sheath, called the axon, may start to breakdown well before the myelin does. In addition, studies have connected demyelination to Alzheimer's and Parkinson's and other neurodegenerative diseases implying that it is part of the general neurodegenerative process rather than the cause. Lastly, viruses tend to attack tissues and cause them to breakdown from the outside in. The nerves in MS, on the other hand, tend to breakdown from the inside out."
From;
http://www.upright-health.com/multiple-sclerosis.html
And;
"It is easy to see how violent venous back jets can be a source of destruction for delicate nerves located near the largest dural sinuses. Reflux can stretch and compress tissues in the brain, possibly to the breaking point. In fact, some researchers have suggested that even edema, that is swelling, can be sufficient to damage nerves due to over stretching them.
Cardiorespiratory waves as a source of venous reflux and nerve damage is a bit more of a stretch for me to accept even if the patient does have incompetant valves. Early on in my research I looked into bats, giraffes and whales because of the extreme inversion flows the brain contends with during head inversion and deep dives. My investigation led me to conclude that these mammals have developed compensatory mechanisms to deal with normal, albeit extreme, inversion flows. If extreme back pressure was the cause then competition weight lifters, trumpet players, gymnasts, yoga masters and jet pilots would most likely have a higher incidence of multiple sclerosis.
Venous back jets like chronic cerebrospinal venous insufficiency (CCSVI) also fails to explain the gender, racial and geographical distribution differences in the incidence of MS. Nonetheless,venous back jets make the most sense when it comes to the likely role of trauma, especially whiplash type trauma, in the cause of MS lesions.
In contrast to venous back jets, my theory as to the destruction of nerves and myelin suggests that chronic craniocervical venous back pressure may be the problem. Furthermore, chronic craniocervical hypertension can decrease CSF pulsatility causing it to accumulate in the brain. It also proposes an explanation for the gender, racial and geographical distribution differences in the incidence of MS. For further information see the section on chronic craniocervical venous back pressure (CCVBP)."
From;
http://www.upright-health.com/backjets.html
And to see the finale go to;
http://uprightdoctor.wordpress.com/2010 ... -or-ccvbp/
"Chiari Conditions, CCSVI and Nerve Compression
Chiari conditions can compress these and other cranial nerves just like the optic nerve mentioned above. Among other things, compression of the hypoglossal nerve can cause slurred speach. Compression of the vagus nerve can cause nausea, vomiting and feelings of fainting. Higher up in the cranial vault, compression of the optic nerve causes pain and blindness. Among other things, nerves do not like compression. Picture hitting your not so “funny” elbow bone and recall what that feels like. It’s like pressing on your eyeball or pinching your skin. It hurts. Nerves are meant to be highly sensitive. It’s there job to be sensitive. That’s why the brain is surrounded by water to protect it from compression against the hard walls of the cranial vault, as well as from itself."
"Because of this close arrangement it only takes microplastic, miniscule misalignment type strains to cause compression of the soft tissues surrounding the openings in the base of the skull and subsequent back pressure against their veins that can have a major impact on fluid mechanics in the brain."
"Back pressure against the veins of the brain near the base of the skull can cause venous congestion called edema inside the brain. It can also decrease the passive CSF production pressure gradient used during upright posture for brain support."
"After you understand the arrangement of these arteries and veins it will be easy to see how small misalignments in the upper cervical spine can have a major impact on the health of the brain. They can also affect the spinal cord, but we aren’t even close to covering issues related to the cord at this juncture. The brain is complicated enough for now."
Thanks Dr.
