Discovery DSALUD. Número 21. Number 21. Octubre de 2000 . October 2000.
Solution for some degenerative central nervous system.
Noda - is a perfect example. They correspond to the English expression Cerebellar Thoracic Outlet Syndrome and refers to pathological compression of the neurovascular structures that sometimes occur at the base of the neck. I will try to explain to the reader the simplest way possible: on both sides of the neck are two regions called the scalene triangle that is bounded by the scalenus anterior, scalenus medius and the rib (see picture). And in between are the subclavian artery, the brachial plexus and the vertebral arteries and internal mammary. Well, sometimes there is a neurovascular compression of these structures causing a malfunction of the nervous structures.
So much that most doctors know. But what these doctors do not seem to know and what Fernandez Noda discovered was that this compression also affects the vertebral artery causing a decreased blood flow to the brain and hindering venous return in the cranial area, causing many of the degenerative processes of the central nervous system, Parkinson's, multiple sclerosis, cerebellar ataxia, epilepsy and some cases of Alzheimer's, among others.
I took the liberty of shortening the quote and correcting the translation on the bottom half.
The point I want to make is that, as you noticed, only arteries are mentioned; the reason is that few years ago nobody paid attention to the veins. But, as you know, in our body any artery is accompanied by a vein. The other point is that if you have external pressure, as the one applied by an hypertrophic muscle, the veins are going to be affected much more then the arteries due to their lower pressure and higher flexibility.
So my contention is that Dr. Noda, with his surgery, was improving the venous return, specially from the vertebral vein, and that is the real reason MS patients improved so much after his surgery.
Dear Dr. Sclafani,
I am posting this because I think it is the missing link.
What I mean is that CCSVI primarily addresses problem with the Internal Jugular Vein and Azygous Vein while CTOS/CTNVS relates mostly to Vertebral Artery/Vein problem.
Dr. Noda operated on more then a thousand patients with impressive results even before CCSVI was in our vocabulary.
I am proposing the two conditions are complementary and this will improve even further our understanding of blood flow impairment related to MS.
This link has an English translation of some of his work: http://health.groups.yahoo.com/group/healingparkinsons/message/870
A special thank to you for going through what we trow at you which require a significant amount of time and dedication on your part.
I am sure everybody here is aware of this and we all are very appreciative of your efforts.
nunzio, i am appreciative of your efforts too. Teach me more. How does one recognize CTOS clinically?
To diagnose this syndrome, we do a complete medical history and the four diagnostic maneuvers, the Adson, hyperabduction, hyperextension and a new maneuver we have developed.
In addition to the above, we use the following diagnostic studies:
3. Imaging of the spine and thorax
4. Doppler ultrasonography with regular exercises and our maneuver in the upper extremities
5. Electromyography (EMG)
6. Study of nerve conduction velocity (NCV)
We added the following diagnostic studies:
7. Somatosensory evoked potentials of short latency of the upper limbs (SEP), which we identified as a specific diagnostic tests in the diagnosis of SPD due to the alteration of P14 waves caused by lack of oxygen and irrigation to the putamen and cortex brain, since 1984
8. Digital Intravenous subtraction Angiography (IVDSA) head and neck, since 1985
9. Possitron Emission Tomography (PET) scan for patients with Parkinson's disease since 1985
10. Single Photo Emission Computed Tomography (SPECT Brain), since 1990 (Table 3)
11. Transcranial Doppler ultrasonography with our maneuver, since 1994
Symptoms produced by CTOS
Headaches (migraine), neck, chest and arm numbness, chest, dyspnea transient memory deficits, dysphagia, dizziness, tinnitus, urinary incontinence, slurred speech, loss of consciousness, paralysis ipsilateral, severe stress, joint temporomandibular, amaurosis fugax, tachycardia, dysmenorrhea, heavy bleeding during menstruation, paresis, snoring and others.
Symptoms caused by Parkinson
Tremors, impaired writing, sialorrhea, seborrhea, shuffling gait, chorea, rigidity, aspect of monkey, and sexual impairment.
A. Described by other authors
12. Aneurysm of the subclavian and vertebral arteries
B. Described by the author (11)
14. Ipsilateral paralysis
15. Loss of temporary or permanent vision
16. Full compression of the subclavian artery with impending gangrene of the upper extremities
17. Symptomatic Parkinson's Disease
18. Early Alzheimer's Disease
19. Pulmonary complications
20. Alzheimer's Disease Functional
22. Multiple Sclerosis
23. Psychological disturbances due to hypoxia
24. Parkinsonism hemodynamic
25. Symptomatic early Parkinson's disease (ESPD)
This was taken from an english translation of a full paper by Dr. Noda.
The link is below
Interestingly they used neck doppler and intacranial doppler