If you have MS, read on as you might want to look into this !
The above article Studied 900 Patients. Results showed 90% of Lyme patients have Hypercoagulation compared to 5% of the General Population. FYI, there are many more articles like the above out there.
I will start on Lovenox for 3 weeks post CCSVI repair. I will also be on Boluoke concomitantly. More on Boluoke below, but please read all of this:
Let me premise my case so you all understand where I am coming from. I have had Neuro Lyme x 4 years. Prior to this, I was a healthy 38 y.o. RN, Married with 2 kids, Nice house, Active, Great Health, etc. My symptoms began as I'm sure many of you can relate after a very stressful event. I lost my best friend in the world to Lung Cancer, my brother.
The first 2 years passed and I had no idea what was wrong with me. Then I had 2 positive Lyme bands through Igenix Labs in Palo Alto, CA. At first I though aha, a month of Antibiotics and I'll be all better. Well research on Lyme and you will see I was dead wrong.
The more I treated, the worse I got. It felt like the Abx were being trapped in my head and I was having what my Lyme Dr. called Brain Herxes. A Herxheimer reaction was first seen in treating Syphilis. The definition is a worsening of symptoms and perhaps the onset on new symptoms. Some theories propose the Toxins being killed off create a temporary inflammatory response from Cytokine release.
When I treat my Lyme, I cry like a 2 year old for days on end, in total despair. Many of my other head symptoms flare as well. Some sort of Anxiety that I call "A plugged in a 110 volt outlet" sensation also goes haywire. This doesn't respond to Xanax or other Benzos, as it's more of a CNS invasion than a true Anxiety. The crying stuff sure feels like the deepest depression one could have when it's going on, but if I hold treatment, it goes away over a week or so depending on what I've taken to treat my Lyme. I get this same effect using Alternative herbs and other Alternative methods such as Colloidal Silver. So, this would rule out any toxic effect from a drug.
Trying to keep this simple, but a little more background on Lyme as it relates to CCSVI. Many of us Lymies are now finding we have CCSVI. The MS society was the first to really jump on this b/c Dr. Z's wife had MS. The Lyme community is sold on the fact that MS is simply a case of Lyme, as is CFS, FMS/ME, etc. I'm not here to debate this, but I do think it is important to point out all of the facts !
With that said, my research for the past 2 1/2 years has been on Lyme. I have been hospitalized 5 times due to my complications with my own case. Lots of blood draws and re-draws as I was told the specimen Hemolized. As an RN, I have come across this from time to time with my patients, but never the amount of times this has happened to me. I thought because my PT/PTT/INR were all ok, that my clotting was ok. My Platelet counts were also normal, as was my D-dimers. In other worse, I had no concerns of having a clotting problem.
So, I had heard of this Fibrinogen talk some time ago, but never gave it much thought until recently. When I found out I had CCSVI recently and read all of your stories about restenosis, thrombosis, etc. despite being on blood thinners, I began to think I might be onto something. Read below and you will see how Boluoke has no effect on PT/PTT, INR, etc.
Boluoke® (lumbrokinase) is the only fully researched oral fibrinolytic supplement on the market. Besides having in vitro studies, animal studies, toxicity studies, and pharmacokinetic studies done, Boluoke® has also been put through all phases of clinical trials (including randomized double blind controlled studies) in China. Lumbrokinase has been studied as a treatment for various clinical conditions, including acute, sub-acute, and chronic conditions that are associated with the presence of hypercoagulation and hypoperfusion.
Lumbrokinase is a more potent enzyme preparation than nattokinase. One of the advantages of Boluoke® (lumbrokinase) is that it does not interfere with the clotting cascade, but instead, works by reducing fibrinogen and fibrin. Boluoke® does not affect INR or aPTT, thus is compatible with Coumadin® or heparin.
I encourage all of you to read into this further and please do share your thoughts here on this thread. I do not want to have this procedure more then once. I hope my Theory is correct even though I'm using the Theory of others, but applying it to CCSVI.
To read more on Boluoke, check out the link below.
Gary Engelman, BSN, RN