This Is MS Multiple Sclerosis Community: Knowledge & Support

Professor Zohara Sternberg of the Jacobs Neurological Institute of Buffalo NY .... has given us something of great value in understanding CCSVI. Please read .... and if you have to .... reread her Abstract listed above . It's that important.

The Abstract is written in true doctor-speak .... so MrSuccess will attempt to explain it in plain talk.

Professor Sternberg is telling us that once a vein is collapsed - whereby the inner walls of the vein come in contact - they cannot disengage from each other .

I agree.

The reason why : SURFACE TENSION ...... MrSuccess would add to this ... .. a created VACUUM also.

CCSVI is the simple concept of a fluid [ BLOOD ] disrupted from returning back to a fluid pump [ your HEART ] . A circuit - heart to head ->>> head back to heart .... is not normal.

Now let me demonstrate the Power of Surface Tension AND the power of a VACUUM.

First: Surface Tension. The most easy example I can give you is the adhesive quality's of two EQUALLY smooth pieces of glass. Slightly moisten both pieces ..... now SLIDE them together .... so that they are exactly one on top of the other . Now try to PULL them apart.

Second : The Power of a VACUUM . Mr.Success offers the " Magdeburg Spheres ".

Professor Sternberg tells us something VERY IMPORTANT .... and that is this .... in the condition CCSVI ..... your heart - a simple diaphram pump - CANNOT create enough fluid FORCE to overcome the collapsed veins .

MrSuccess asks ..... Is CCSVI perhaps truly .....a HEART CONDITION ?

Is the heart now creating a VACUUM in the vein returning blood from the brain to the heart ?

DO NOT underestimate the power of surface tension or a vacuum . Dr. Sternberg clearly understands this.


MrSuccess

One of the best things about HRV is that it’s an incredibly simple and non-invasive test. All you will have to do is stay in a stable, resting position for 2:30 while the BioForce HRV records the intervals between your heart beats. You can even do it while laying in bed in the morning.

MrSuccess
i would disagree with the statements that once vein walls touch they cannot separate. i see it all the time, where veins collapse and then expand. it is a natural phenomenon.

it is most apparent in patients who have compression stenoses, such as at the skull base, at C1-2, in J2 with muscle compressions, with the may thurner syndrome and the nutcracker phenomenon. it is clearly proven by viewing the vein in IVUS

DrS

Hi Dr.S - You are the "inside guy " when it comes to CCSVI. It appears there may be some growth in this area [ IVUS ] with other professionals now using it.

IVUS , if I understand it correctly , actually FORCES open collapsed veins as it is guided through pwMS . I suppose webs and flaps and other inner workings .... get plowed through also .

Here is what I don't get .... upon REMOVAL of the IVUS ...... How do you know for sure that the vein has not immediatley returned to a full or partial closure ?

Stents appear to be the solution . However , their composition [ metal ] seems to be the devil in the details ...... the body wants to reject them ..... hence a lifetime on drugs ...

Q : Do you think it possible that stents can be produced using ones own body parts ? I threw this suggestion out a few years ago , that something like the plaque on our teeth could be removed , moulded into stents , and then used in veins.

As it is already naturally produced in your body ....... rejection should not be an isssue ? Nice little tubes of natures own concrete .....

MrSuccess

Neurology. 2003 Sep 23;61(6):851-3.
Fatigue in MS is related to sympathetic vasomotor dysfunction.
Flachenecker P, Rufer A, Bihler I, Hippel C, Reiners K, Toyka KV, Kesselring J.
Source
Department of Neurology, Julius-Maximilians-Universität, Würzburg, Germany.
Abstract
The authors studied standard autonomic function tests and measures of heart rate variability in 60 patients with multiple sclerosis (MS) and correlated results with the Fatigue Severity Scale and the Modified Fatigue Impact Scale. The authors found that autonomic responses correlated with fatigue resembling a hypoadrenergic orthostatic response, possibly due to a sympathetic vasomotor lesion with intact vagal heart control. Treatments to control sympathetic dysfunction for MS-associated fatigue deserve further study.

Autonomic dysfunction in Guillain-Barré syndrome and multiple sclerosis.
Flachenecker P.
Source
Neurological Rehabilitation Center "Quellenhof", Kuranlagenallee 2, 75323, Bad Wildbad, Germany.
Erratum in
J Neurol. 2008 Feb;255(2):309-10.
Abstract
This review gives an overview of autonomic dysfunction encountered in Guillain-Barré syndrome (GBS) and multiple sclerosis (MS). In GBS, cardiovascular dysregulation is common and may lead to serious bradyarrhythmias that need to be recognised for the early initiation of appropriate therapy. Although standardised autonomic tests were useful for the diagnosis of autonomic failure, they were not able to indicate vagal over-reactivity. In this regard, eyeball pressure testing may correctly identify patients at risk for impending and potentially life-threatening bradyarrhythmias which may easily be administered at the bedside. In MS, cardiovascular autonomic dysfunction is usually of minor clinical importance. However, orthostatic intolerance may be present in approximately 50% of patients and could easily be detected by routine measurements of heart rate and blood pressure during rest and during standing. More subtle alterations may require more sophisticated methods such as autonomic reflex testing or baroreflex stimulation. Several in vitro, animal and clinical studies provide evidence that there are many interactions between the sympathetic nervous system and the immune system giving rise to the hypothesis that autonomic dysfunction in MS may not only be a consequence of the disease, but may in itself affect the course of MS.