A forum to discuss Chronic Cerebrospinal Venous Insufficiency and its relationship to Multiple Sclerosis.


Postby 1eye » Wed Sep 10, 2014 4:56 am

Address of the person responsible for updating the info.:
"Try - Just A Little Bit Harder" - Janis Joplin
CCSVI procedure Albany Aug 2010
'MS' is over - if you want it
Patients sans/without patience

I am not a doctor. Do not take anything I say as medical advice.
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Postby CureOrBust » Fri Nov 21, 2014 2:11 am ... -patients/

However, while the debate over CCSVI and MS continues, new data suggests that Dr. Zamboni may have gotten it right in looking at the veins for treatment, but it might not be venous abnormalities that are the real problem, with a team of California-based researchers contending that it is the nerves surrounding the veins not the veins themselves that are being treated by ballooning, with expansion of the jugular vein leading to stimulation of the autonomic nerve fibers, which run alongside the jugular and are responsible for communication between the brain and the central nervous system. They say that ongoing research and a growing body of clinical data strongly indicate that Dr. Zamboni’s CCSVI procedure may be the first viable treatment for an even more pervasive problem — dysautonomia — which is seen not only in almost all MS patients but also in patients diagnosed with a long list of other diseases and conditions. As a result, they believe the CCSVI procedure is better described by the term TVAM (Transvascular Autonomic Modulation).

Transvascular Autonomic Modulation: A Modified Balloon Angioplasty Technique for the Treatment of Autonomic Dysfunction in Multiple Sclerosis Patients
Conclusion: The combination of balloon angioplasty of anatomically normal veins coupled with external compression during dilation of these veins can improve indicators of ANS dysfunction. The safety and efficacy of TVAM in MS patients observed in this pilot study is encouraging, paving the way for the treatment of dysautonomia in pathological states other than MS. Further studies should investigate TVAM in a larger MS cohort.
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Postby SammyJo » Tue Feb 16, 2016 12:12 pm

I was searching Alzheimer's Disease, and I was delighted to see CCSVI noted, with the statement

"It is possible that this is also the cause of neurodegeneration in AD. "

Which followed a summary of CCSVI in MS "This theory holds that increased venous pressure could be the main reason for the formation of colloids, lymphocyte and erythrocyte extravasation, promoting an inflammatory reaction in the brain and spine in cases of MS"

Then they put together a nice table, 8 CCSVI papers by 7 different authors, using 7 different measurement techniques.

Interesting to see that someone from outside the USA neurology lockbox, an author in Kenya from another neuro disease specialty, appears to find the CCSVI MS theory perfectely plausible, and supportive of one of the theories on AD. He also summarizes the history of theories on AD. That's how MS researchers should approach it, that CCSVI is a new and plausible theory that needs to be lined up against the old theories.

It's been 7 years. Are we there yet? Are there any countires yet where CCSVI treatment is now a normal practice of medicine for MS?

Hope all of us CCSVI-ers are doing well!

A Current Understanding of Alzheimer's Disease and the Prospects of Phytopharmacological Intervention as a Management Strategy.

Journal: Juma, J Neurol Disord 2015, 3:4
Author Juma KK*
Department of Biochemistry and Biotechnology, Kenyatta University, Nairobi, P.O. Box 43844-00100, Kenya ... 000244.pdf
RRMS '95 SPMS '02 | CCSVI 10/09 | Adult stem cells 2012 |
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Postby jimmylegs » Thu Feb 16, 2017 9:20 pm

i'm out of the loop. thoughts on these?

Extracranial Venous abnormalities: A true pathological finding in patients with multiple sclerosis or an anatomical variant? ... 016-4314-6

Impaired Neurovisceral Integration of Cardiovascular Modulation Contributes to Multiple Sclerosis Morbidities ... 015-9599-y

Venous compression syndrome of internal jugular veins prevalence in patients with multiple sclerosis and chronic cerebro-spinal venous insufficiency.
odd sx? no dx? check w/ dietitian
99% don't meet these. meds/lifestyle can affect levels
status can be low in ms & other cond'ns
'but my results are normal'. typical panels don't test all
deficits occur in 'normal' range
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Postby AMcG » Mon Feb 20, 2017 4:18 am

"• The venous anatomy of the neck in patients without MS demonstrates multiple variants

• Asymmetry and stenoses of the internal jugular veins are common

• Collateral neck veins are not uncommon in patients without MS

• These findings do not support the theory of chronic cerebrospinal venous insufficiency"

This is an obvious non-sequitur that has been stated many times before. Because there is wide variation in normal people (as we all know) it does not follow that no variation can possibly be pathological. It just means it is necessary to be able to identify what is and what is not pathological variation.

If you look at maps of city center streets in any country you will find the pattern of streets shows infinite variation (far greater than you find in peoples' necks.) Does it therefore follow that traffic jams are impossible? Clearly nonsense.
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