Zinc deficiency – Zinc is necessary in maintaining intestinal wall integrity. Supplementing with zinc could contribute significantly to healing a leaky gut in about eight weeks (Sturniolo 2001). Zinc is also instrumental in a maintaining a healthy immune system (Prasad 2002). The synthesis of serotonin involves zinc. Since serotonin is also necessary for melatonin synthesis, a zinc deficiency may result in low levels of both of these compounds, causing problems with the sleep cycle, calming, and hyperness.
Malnourished children have thymic atrophy which is reversed by zinc supplementation. To see if their defect in cell-mediated immunity was also associated with zinc deficiency ten children were skin-tested with Candida antigen on both arms. One test site was covered with local zinc sulphate and the other with placebo ointment. There was a highly significant increase in the typical delayed-hypersensitivity reaction at the site covered with zinc. The magnitude of the difference between the supplemented and unsupplemented arms correlated negatively with the plasma-zinc concentration. These data show that zinc deficiency is a cause of the immunoincompetence seen in malnutrition. The normal reactions of the zinc-supplemented side indicate that, of the many nutritional deficits of malnourished children, zinc deficiency specifically impairs the cell-mediated immune system. Local skintesting with and without zinc may provide a measure of zinc status. Local application of zinc may enhance the reliability of tests to diagnose diseases such as tuberculosis in malnourished patients.
Zinc metabolism is closely related to Candida because 1) the zinc/copper balance is critical, and 2) zinc is required for many essential enzyme systems, including production of digestive enzymes and synthesis of all body proteins.
A zinc imbalance is indicated on a tissue mineral chart by a zinc level less than 12.0 mgs/% or greater than 20.0 mgs/%, or a zinc/copper ratio greater than 12.0 mgs/%. A phosphorus level greater than 16 or less than 12 may also indicate a zinc imbalance.
Deficiency of zinc is common for several reasons:
• Use of superphosphate fertilizers and hybrid crops have contributed to widespread zinc deficiency in all foods.
• Processing and refining further depletes foods of their zinc content. For example, zinc loss occurs in the conversion of whole wheat to white flour, in the conversion of sugar cane to white sugar, and in spraying of frozen and canned vegetables with EDTA to retain color.
• Foods, relatively low in zinc, such as chicken and fish are being increasingly substituted for higher-zinc foods such as beef and red meats. Soy protein, commonly substituted for beef, is low in zinc.
• Stress of any type results in zinc depletion.
• Zinc deficiency is accentuated if copper exposure is high, because of a copper-zinc antagonism. Copper exposure is higher today for several reasons:
– Birth control pills raise tissue copper levels by raising estrogen levels.
– Copper is absorbed from the Copper-7 intrauterine device.
– Water remaining in copper pipes, and consumption of high-copper foods such as soy, avocado, and chocolate are sources of copper.
– Stress causes copper levels to increase, by causing a zinc deficiency.
Phytate concentrations of the unleavened flat bread (tanok or lavosh) prepared from high extraction wheaten meal in Iranian villages are much greater than those of leavened flat breads (bazari or sangak) made in commercial bakeries in cities of the same region. As a result, the phytate intake of the rural population considerably exceeds that of the urban population. Excessive intakes of phytate may explain the occurrence of overt zinc deficiency among villagers and its absence in the cities.
Leonard wrote:The relationship between MS and Vitamin D is well known. My exacerbations always occured in the Spring, typically April or May, when the Vitamin D level was low. Now, it appears that the link is established through the gut bacteria! Whow, that is big! What is the case?
Sufficient serum 25OHD levels allow cells in the body to synthesize cathelicidin when microbiota need to be killed.
The fact that there is this interaction between Vitamin D and the gut bacteria is very powerful and further strengthens the two stage disease concept (ccsvi/bacteria) as described in the above posting.
Hence we may now conclude (see also my earlier posting above):
The narrowings can be treated by angioplasty.
The segmented filamentous bacteria, that originate from the gut, can be treated by antibiotics and by maintaining a sufficienty level of Vitamin D. Also diet (low fat/low sugar; Swank-like) or fasting may help: see http://www.thisisms.com/ftopict-17679.html
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