In the next few years, research in several promising areas is likely to bear fruit, especially:
- an improved understanding of how best to apply treatments that modify the immune response and to limit the effects of MS,
- the implementation of neuroprotective strategies, including the MS Society’s trial of lamotrigine, to prevent disability from occurring, and
- progress on how to repair the damaged nervous system in MS to help with recovery from established disability.
I truly believe they are as well.better times are getting nearer
CONCLUSIONS: In this experimental SIV model of HIV CNS disease, minocycline reduced the severity of encephalitis, suppressed viral load in the brain, and decreased the expression of CNS inflammatory markers. In vitro, minocycline inhibited SIV and HIV replication. These findings suggest that minocycline, a safe, inexpensive, and readily available antibiotic should be investigated as an anti-HIV therapeutic.
Experimental infection of neonatal mice with neurotropic reoviruses is a classic model for studying the pathogenesis of viral encephalitis.
These findings, similar to those reported for neurodegenerative diseases, indicate that minocycline does not prevent development of fatal reovirus encephalitis but delays disease onset and progression, suggesting that minocycline treatment may provide a useful adjunctive therapy in viral CNS infections.
Minai wrote:he wants me to take an herbal concoction in capsules. Starting off slowly with it, but increasing it to aggressive levels. He takes it himself to ward off viruses ... I told him I'd try it, but only after I looked it up to see what it has in it
Minai wrote:But, since viruses are anaerobes, maybe this anemia created just the environment the EBV needed to thrive
Minai wrote:Will PM the URL to you.
Minai wrote:but being the skeptic that I am about such things; my expectations are low
This defective innate immune response was also associated with a widespread viral replication and neurodegeneration....
These data provide evidence that interaction of E(2) with their nuclear ERalpha plays a critical role in the control of cytokines involved in the transfer from the innate to adaptive immunity. mice lacking E(2), ... allows pathogens to hide from immune surveillance and exacerbates neuronal damages during viral encephalitis.
E(2) replacement therapy totally rescued the ability of the endotoxin to trigger microglial cells and these permissive effects of E(2) are mediated via the estrogen receptor (ER)alpha.
Just another reason to think about balanced hormone levels.
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