hi again!
re neuropathy (can you describe further, as specifically as possible?):
here's a great resource that looks (in specific and technical detail) at some neuropathies associated with nutritional deficiencies:
http://neuromuscular.wustl.edu/nother/vitamin.htmfor several of the vitamins and minerals listed, there happens to be research investigating their status in ms patients. so with a few blood tests you can find out if you match the nutritional profile of an ms patient, or the profile of a healthy person.
(there's also chromium deficiency neuropathy which is not listed at first glance, but research is out there on that one too. and likely others. but it's a good starting point)
depression - some links to b complex vitamins (depression is mentioned in the syndromes site above, under vit b6), magnesium, and vitamin d3:
Folic acid deficiency and depression
http://www.sciencedirect.com/science/ar ... 8280735867Depression and magnesium deficiency.
http://www.ncbi.nlm.nih.gov/pubmed/2722406Quote:
The psychiatric symptoms of magnesium deficiency are unspecific, ranging from apathy to psychosis, and may be attributed to other disease processes associated with poor intake, defect absorption, or excretion of magnesium. Serum magnesium should be determined when there are symptoms consistent with magnesium deficiency and/or in conditions which can lead to a deficiency, e.g., malabsorption, malnutrition, alcoholism and diuretic treatment. A low serum value suggests magnesium deficiency, but the diagnosis is reinforced with analyses of magnesium in the urine and a loading test with magnesium. Magnesium can be given orally or intramuscular/intravenously.
Vitamin D deficiency may play a role in depression
http://www.medical-hypotheses.com/article/S0306-9877(07)00240-X/abstract
Quote:
Vitamin D is known to be widely deficient in Western populations. The implications of this in terms of bone health are increasingly understood, yet its impact on other health areas, particularly mental health, is unclear. Recent data suggests that hypovitaminosis D may be common, especially in the elderly. Other studies have suggested that low levels of vitamin D are associated with poor mood. There are a number of trials that have suggested a role for Vitamin D in the supplementary treatment of depression. Dose may be a critical issue, as sun exposure and dietary intake may be low and high doses may be required.
i'd try magnesium for anxiety too, it's pretty awesome
A Synergistic Effect of a Daily Supplement for 1 Month of 200 mg Magnesium plus 50 mg Vitamin B6 for the Relief of Anxiety-Related Premenstrual Symptoms: A Randomized, Double-Blind, Crossover Study
http://online.liebertpub.com/doi/abs/10 ... 0900318623magnesium's also good for fatigue, muscle pain and twitches, headaches, constipation (if you use the right form eg oxide - for the other problems you use a different form)...
i used to have eye floaters and don't have them any longer, but i have no idea which nutrient or combination of nutrients helped!
my best recovery from serious brain fog was repletion with adequate zinc. i think it's likely to help with the alopecia as well. i suggested to one of my co-workers that zinc would help her conceive, and at the time i didn't realize she had shaved her head due to alopecia. i told her to take zinc (and magnesium just to help with any possible cramps etc in the meantime) and she was pregnant in no time. has a full head of hair again too
i looked up livedo reticular in combination with deficiency, and the results go straight to APS and a deficiency of protein C:
Acquired protein C deficiency in a patient with primary antiphospholipid syndrome. Relationship to reactivity of anticardiolipin antibody with thrombomodulin
http://ukpmc.ac.uk/abstract/MED/2542543Quote:
A 16-year-old boy had recurrent venous thromboses and pulmonary thromboembolism that caused him pulmonary hypertension. He also had livedo reticularis, thrombocytopenia and high titer IgG antiphospholipid (cardiolipin) antibodies. In the absence of clinical and laboratory evidence of SLE, he was considered to have a primary antiphospholipid syndrome. Coagulation studies revealed a functional deficiency of protein C although it was present in normal antigenic amounts. Since both his parents had normal functional and antigenic protein C findings, his deficiency was considered acquired...
i don't have time to dig too deep today, but i'm hypothesizing there's a zinc-dependent pathway somewhere in the mix, for regulating protein C.
that's all for now! will find that link i mentioned above too.
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