many nutrients seen to be low in ms patients, are important for vascular health.
"Galvanising forces in vascular health and disease: is dietary zinc protective? "
"Magnesium and vascular tone and reactivity"
"The Vitamin D Epidemic and its Health Consequences..There is mounting scientific evidence that implicates vitamin D deficiency with an increased risk of type I diabetes, multiple sclerosis, rheumatoid arthritis, hypertension, cardiovascular heart disease, and many common deadly cancers."
"C-reactive protein, dietary n-3 fatty acids, and the extent of coronary artery disease"
"Importance of both folic acid and vitamin B12 in reduction of risk of vascular disease"
there's not much that can be done to stop a jugular valve from recoiling if it wants to recoil.
In my wife's case, it seems that her jugular veins is still open, after 3 years - at least symptoms have been away. Could it be possible that healthy life style.. nutrition and hard exercise might be related factors?
Magnesium ions control prostaglandin reactivity of venous smooth muscle from spontaneously hypertensive rats.we had some chats about magnesium a couple years ago, may be of interest:
Cece wrote:With angioplasty patients, if angioplasty was needed because of fatty deposits in the arteries, then it makes a lot of sense to eat less fat. With venoplasty ccsvi patients, there are no fatty deposits in the veins. It's a big distinction between the two, I think, and it's one way in which what's known about angioplasty in heart disease patients can't carry over directly as knowledge about CCSVI patients.
Long-term exercise increases the expression of NOS in the vessel wall and augments endothelium-dependent vasodilation.20 21 In addition, both nNOS and eNOS are elevated in skeletal muscle by exercise. The article by Kojda and colleagues22 in the current issue of Circulation confirms the effect of long-term exercise on eNOS expression. This effect of exercise may be mediated by shear stress. When endothelial cells are exposed to fluid flow in culture, NOS mRNA increases.23 This increased expression of eNOS in response to fluid flow may be mediated by shear stress and/or oxidant responsive elements in the promoter region of the gene.24 25 The increased expression of NOS in the vessel wall has physiological effects. Endothelium-dependent vasodilation is enhanced by long-term exercise.19 20 Furthermore, in individuals with impaired endothelium-dependent vasodilation (as in heart failure), exercise restores endothelial response.26
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