Chronic Cerebrospinal Venous Insufficiency (CCSVI)-

skydog · Post by **skydog** » Sun Mar 22, 2009 8:50 am

Andrew Great info… Understand your concern about the inversions. My thought is the combination of a good nights sleep ( eight hrs inclined ) then twenty minutes on the inversion table at the end of the day gently stimulates the whole system. I use my inversion table more for the stretching than inverting. Also I rarely go for full inversion. What seems to feel best is balancing flat for a couple of minutes to relax then by raising one arm at a time going to 70 degrees inverted. Breath come back to a slightly positive. I alternate this for five reps. Relax flat to slightly positive for a minute. I repeat this for 20 minutes. Meditate and go about the evening. Your inclined bed is working wonders for both my wife and I. She used to snore quite loudly. Now she purrs. More restful sleep for both of us. Peace Mark

SarahLonglands · Post by **SarahLonglands** » Sun Mar 22, 2009 9:21 am

AKF, I asked you before but you didn't answer: have you had a reply from Prof Zamboni yet?

Sarah

AndrewKFletcher · Post by **AndrewKFletcher** » Sun Mar 22, 2009 11:07 am

No reply as of yet Sarah.

SarahLonglands · Post by **SarahLonglands** » Mon Mar 23, 2009 4:14 am

Well I never.........

Unlike other people, to mention no names.

AndrewKFletcher · Post by **AndrewKFletcher** » Mon Mar 23, 2009 6:31 am

Can you elaborate?

Anecdote wrote:Well I never.........

Unlike other people, to mention no names.

SarahLonglands · Post by **SarahLonglands** » Mon Mar 23, 2009 6:41 am

No, sorry, its private.

Post by **jimmylegs** » Mon Mar 23, 2009 7:14 am

Loobie · Post by **Loobie** » Mon Mar 23, 2009 12:39 pm

ditto

Artifishual · Post by **Artifishual** » Mon Mar 23, 2009 5:44 pm

what did i miss!?!?!?! somebody PLEASE

oh yeah homeskillet, you owe me $75 for a new pair of fishing pants.......just say no to Mg.

I will not elaborate either

cheerleader · Post by **cheerleader** » Mon Mar 23, 2009 5:50 pm

Just some goofy squabbling, Arti. You haven't missed anything real important.
WHOA! Guess that mag got things moving?

Don't be blamin' legs.
AC

Artifishual · Post by **Artifishual** » Mon Mar 23, 2009 5:54 pm

you could say so, hey she is the big proponent of mg. i think she said something like 500 mg a day?! and i was on top of a huge bridge, about 600 feet in the air.

catfreak · Post by **catfreak** » Mon Mar 23, 2009 6:06 pm

The mag just does not do this to me

I wish it did

Arti, I think you must have really NOT been in a bind!

CF

gibbledygook · Post by **gibbledygook** » Tue Mar 24, 2009 2:52 am

Do you use magnesium citrate? At Vivamayr clinic in Austria they give you magnesium citrate powder with potassium and you have to drink it every few hours. This clinic is legendary for the bowel clearance!!

I also find capsaicin useful in keeping me regular and prefer it to magnesium as long as I'm not having a relapse as it is somewhat vasoactive and may irritate the lesion if the vein wall is breached. I think!

gibbledygook · Post by **gibbledygook** » Tue Mar 24, 2009 4:39 am

I didn't realize this until today but most cheap baking sodas contain aluminium. I think I'll have to start baking my own bread

1: Neurosci Lett. 2008 Nov 7;445(1):42-6. Epub 2008 Sep 3. Links
Effects of acute exposure to aluminum on blood-brain barrier and the protection of zinc.Song Y, Xue Y, Liu X, Wang P, Liu L.
Department of Experimental Center of the Functional Subjects, College of Basic Medicine, China Medical University, Shenyang 110001, China.

Aluminum and zinc are two important trace elements in an organism. Although several studies have demonstrated their impacts on the intelligence, very little was known about their effects on the integrity of blood-brain barrier (BBB). To study the effects of aluminum and zinc on the permeability of BBB, different doses of aluminum and appropriate zinc were administered to rats. Evans blue was detected in brain to determine the permeability of BBB. The ultrastructure of BBB was observed under the transmission electron microscope. Immunohistochemistry and Western blot method were used to detect the expression of skeleton protein F-actin and tight junction protein occludin in brain capillary endothelium. The data indicated that compared with the control group, Evans blue in brains increased (P < 0.01), the ultrastructure of BBB changed and the expression of F-actin and occludin decreased (P < 0.01) in the aluminum-toxic group. Compared with the aluminum-toxic groups, the permeability of BBB to Evans blue decreased (P < 0.01), the damage of the BBB ultrastructure was attenuated and the expression of F-actin and occludin increased (P < 0.05) in the aluminum-zinc group. Our present studies suggest that aluminum increases the permeability of BBB by changing its ultrastructure and the expression of occludin and F-actin. Zinc can protect the integrity of BBB in juvenile rats that are exposed to aluminum and inhibit the decrease of tight junction protein occludin and F-actin expression in BBB.

PMID: 18786610 [PubMed - in process]

color

1: Mult Scler. 2006 Oct;12(5):533-40. Links
Elevated urinary excretion of aluminium and iron in multiple sclerosis.Exley C, Mamutse G, Korchazhkina O, Pye E, Strekopytov S, Polwart A, Hawkins C.
Birchall Centre for Inorganic Chemistry and Materials Science, Lennard-Jones Laboratories, Keele University, Staffordshire, UK. c.exley@chem.keele.ac.uk

Multiple sclerosis (MS) is a chronic, immune-mediated, demyelinating disease of the central nervous system of as yet unknown aetiology. A consensus of opinion has suggested that the disorder is the result of an interplay between environmental factors and susceptibility genes. We have used a battery of analytical techniques to determine if the urinary excretion of i) markers of oxidative damage; ii) iron and iii) the environmental toxin aluminium and its antagonist, silicon, are altered in relapsing-remitting (RRMS) and secondary progressive MS (SPMS). Urinary concentrations of oxidative biomarkers, MDA and TBARS, were not found to be useful indicators of inflammatory disease in MS. However, urinary concentrations of another potential marker for inflammation and oxidative stress, iron, were significantly increased in SPMS (P<0.01) and insignificantly increased in RRMS (P>0.05). Urinary concentrations of aluminium were also significantly increased in RRMS (P<0.001) and SPMS (P <0.05) such that the levels of aluminium excretion in the former were similar to those observed in individuals undergoing metal chelation therapy. The excretion of silicon was lower in MS and significantly so in SPMS (P<0.05). Increased excretion of iron in urine supported a role for iron dysmetabolism in MS. Levels of urinary aluminium excretion similar to those seen in aluminium intoxication suggested that aluminium may be a hitherto unrecognized environmental factor associated with the aetiology of MS. If aluminium is involved in MS then an increased dietary intake of its natural antagonist, silicon, might be a therapeutic option.

PMID: 17086897 [PubMed - indexed for MEDLINE]

link

I see that aluminium ions mess with the structure of elastin. Aluminium chloride inhibits elastin calcification BUT changes elastin's structure. Elastin is part of the vessel wall.

1: Am J Pathol. 1999 Sep;155(3):973-82. Links
Elastin calcification and its prevention with aluminum chloride pretreatment.Vyavahare N, Ogle M, Schoen FJ, Levy RJ.
Department of Pediatric, Division of Cardiology, Joseph Stokes, Jr. Research Institute, The Children's Hospital of Philadelphia, Philadelphia, Pennsylvania, USA.

Elastin, an abundant structural protein present in the arterial wall, is prone to calcification in a number of disease processes including porcine bioprosthetic heart valve calcification and atherosclerosis. The mechanisms of elastin calcification are not completely elucidated. In the present work, we demonstrated calcification of purified elastin in rat subdermal implants (Ca(2+) = 89.73 +/- 9.84 microgram/mg after 21 days versus control, unimplanted Ca(2+) = 0.16 +/- 0.04 microgram/mg). X-ray diffraction analysis along with resolution enhanced FTIR spectroscopy demonstrated the mineral phase to be a poorly crystalline hydroxyapatite. We investigated the time course of calcification, the effect of glutaraldehyde crosslinking on calcification, and mechanisms of inhibition of elastin calcification by pretreatment with aluminum chloride (AlCl(3)). Glutaraldehyde pretreatment did not affect calcification (Ca(2+) = 89.06 +/- 17.93 microgram/mg for glutaraldehyde crosslinked elastin versus Ca(2+) = 89.73 +/- 9.84 microgram/mg for uncrosslinked elastin). This may be explained by radioactive ((3)H) glutaraldehyde studies showing very low reactivity between glutaraldehyde and elastin. Our results further demonstrated that AlCl(3) pretreatment of elastin led to complete inhibition of elastin calcification using 21-day rat subdermal implants, irrespective of glutaraldehyde crosslinking (Ca(2+) = 0.73-2.15 microgram/mg for AlCl(3) pretreated elastin versus 89.73 +/- 9.84 for untreated elastin). The AlCl(3) pretreatment caused irreversible binding of aluminum ions to elastin, as assessed by atomic emission spectroscopy. Moreover, aluminum ion binding altered the spatial configuration of elastin as shown by circular dichroism (CD), Fourier transform infrared (FTIR), and (13)C nuclear magnetic resonance (NMR) spectroscopy studies, suggesting a net structural change including a reduction in the extent of beta sheet structures and an increase in coil-turn conformations. Thus, it is concluded that purified elastin calcifies in rat subdermal implants, and that the AlCl(3)-pretreated elastin completely resists calcification due to irreversible aluminum ion binding and subsequent structural alterations caused by AlCl(3).

PMID: 10487855 [PubMed - indexed for MEDLINE]

link

Post by **jimmylegs** » Tue Mar 24, 2009 4:49 am

is there baking soda in commercial bread? i don't really do much bread i guess, but baking it is fun

haven't done that in ages..

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Chronic Cerebrospinal Venous Insufficiency (CCSVI)-

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