CCSVI and CCVBP

A forum to discuss Chronic Cerebrospinal Venous Insufficiency and its relationship to Multiple Sclerosis.
wallace
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Re: CCSVI and CCVBP

Post by wallace »

After 28 sessions of Chiro with little subjective improvement I decided to take a break from Chiro treatment to try this therapy.

Too soon after a month of this treatment to comment personally but I find the idea intriguing! So intend to continue. Sometimes I feel blood is nourishing my brain!

I built up gradually raising the bottom of my bed so that your lower torso is higher than your heart. Now bottom of bed up to 12 inches higher.

David Wolfe in his book "Longevity now" says he always sleeps inverted and reports amazing health benefits.

I have an inversion table in storage but using it the past never did much for me and I think this is different but perhaps for me at least more profitable.

Also started a walking regime which I think is a good exercise to combine with this idea.

I will keep you informed of what happens!!
Last edited by wallace on Wed Mar 11, 2015 6:56 am, edited 1 time in total.
wallace
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Re: CCSVI and CCVBP

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I did a quick search on the net about this and didnt find much on it. This is the opposite of Bed incline therapy!!!
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uprightdoc
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Re: CCSVI and CCVBP

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Hello Wallace,

I became very interested in inversion when I started my research. Consequently, I studied bats, whales and giraffes. I also studied microgravity which has similar effects to inversion. I have recommended inversion therapy for certain patients with particular neurological and orthopedic conditions but you have to be careful with MS and other neurodegenerative conditions as well as benign intracranial hypertension. Inversion increases intracranial (brain) and intraocular (eye) pressure. It can also increase pressure/volume/B-waves in the brain which can be destructive. As i mentioned in my book, inversion flows in astronauts will most likely cause glaucoma and hydrocephalus in susceptible individuals. We know now that astronauts are having problems with loss of vision. I would not recommend inversion therapy to anyone with optic neuritis unless they are under professional supervision. I would not recommend inversion therapy to anyone with signs of increased ICP unless they are under professional supervision. There are other problems as well. That said slant board therapy can be useful for relieving circulatory issues in the lower body.
wallace
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Re: CCSVI and CCVBP

Post by wallace »

Thanks . I appreciate the response.

Yes but what about sleeping this way?

Richard Horowitz in his interesting book: why dont i get well says chronic illness is linked to POTS(which ties in very well with your work) but he also has a chapter on sleep and says ill people are not properly sleeping(deep REM sleep etc). The notion that sleeping inverted could restore deep sleep etc seems to me very interesting.


I agree with you on dismissing inclined bed therapy on your blog.
wallace
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Re: CCSVI and CCVBP

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wallace
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Re: CCSVI and CCVBP

Post by wallace »

The ancient Egypians always slept on inclined beds. The question remains which way up were they????????????
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Robnl
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Re: CCSVI and CCVBP

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It's all about flow....
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uprightdoc
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Re: CCSVI and CCVBP

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I disagree with Horowitz. Most chronic illnesses are not related to POTS. Moreover, inversion does nothing physiological to restore deep sleep. It doesn't increase blood flow to the brain. There is no real benefit to the brain. Inversion increases pressure in the superior sagittal sinus. Increased pressure in the superior sagittal sinus decrease cerebral perfusion pressure. Decreased cerebral perfusion pressure decreases brain blood and CSF flow. Decreased CSF flow slows down the removal of destructive elements from the brain such as heavy metals, metabolic wastes, bacteria, viruses, Beta amyloids, alpha and gamma synucleins etc. Inversion also increases pressure in the eye (intraocular pressure). Some astronauts exposed to prolonged inversion flows from microgravity have experienced permanent decreased vision. From my personal and professional experience, inversion isn't that helpful to orthopedic problems either such as disc problems. Aside from that, inversion may have a role in treatment options for certain types of problems related to faulty craniospinal hydrodynamics but I don't have the time to discuss ithe topic here.

I haven't read Horowitz's book on Lyme disease but Lyme disease is old news. There were many doctors in my area who claimed to be the world's leading expert on Lyme disease. Lyme disease treatment was very trendy while I was in practice along with TMJ. Both Lyme disease and TMJ dysfunctio were blamed for a multitude of chronic illnesses from A-Z. Except for acute cases, antibiotic treatment for chronic cases of supposed (possible) Lyme disease based on scanty evidence wasn't effective in any of the cases I saw.
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uprightdoc
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Re: CCSVI and CCVBP

Post by uprightdoc »

It certainly plays an important role.
wallace
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Re: CCSVI and CCVBP

Post by wallace »

Horowitz book is worth perusing.


I am a big fan of Ancient egyptian culture so if they slept on inclined beds then thats good enough recommendation for me!! But doing it inverted makes more sense to me than the normal head up position but if only I had an Ancient egyptian doc to advise...oh well!! They would tell you which way up to be!!

I plan to continue!

But my level bed is now a thing of the past!

Thanks for your response.

I came accross this which doesnt really back me up but I thought it was worth posting
http://www.cjem-online.ca/v6/n1/p48
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uprightdoc
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Re: CCSVI and CCVBP

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The ruling class of ancient Egyptians also performed artificial skull deformation which can cause problems with blood and CSF, as well as hydrocephalus.

The article mentions using the Trandelenberg position for hypotension and shock which I mentioned previously. It also discusses the adverse impact inversion has on respiration and intracranial pressure.

I have recommended slant board therapy to patients for a variety of conditions related to compression, congestion, hypotension and edema in lower body. Further research also needs to be done to determine its usefulness in certain types of neurological disorders that I won't go into here.
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Re: CCSVI and CCVBP

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Abstract
The dominant hypothesis in multiple sclerosis is that it is an autoimmune disease; however, there is considerable evidence that the immune attack on myelin may be secondary to a cytodegenerative event. Furthermore, the immune modulating therapies longest in clinical use, although modulating the frequency and severity of exacerbation, do not affect long-term progression towards disability. Clearly alternative perspectives on the etiology of multiple sclerosis are warranted. In this paper I outline the commonalities between idiopathic normal pressure hydrocephalus and multiple sclerosis. These include decreased intracranial compliance as evidenced by increased cerebrospinal fluid volume and velocity of cerebrospinal fluid flow through the cerebral aqueduct; increased ventricular volume; periventricular demyelination lesions; increase in size of Virchow-Robin spaces; presence of Hakim's triad comprised of locomotory disabilities, cognitive problems and bladder control problems. Furthermore, multiple sclerosis is associated with decreased arterial compliance. These are all suggestive that there is a pulse wave encephalopathy component to multiple sclerosis. There are enough resemblances between normal pressure hydrocephalus and multiple sclerosis to warrant further investigation. Whether decreases in intracranial compliance is a consequence of multiple sclerosis or is a causal factor is unknown. Effective therapies can only be developed when the etiology of the disease is understood.
http://www.ncbi.nlm.nih.gov/pubmed/25760216
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uprightdoc
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Re: CCSVI and CCVBP

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Thanks Nigel. I include Juurlink's paper in the references for my next paper on craniospinal hydrodynamics along with papers from neurosurgeons such as Williams, Oldfield, Gardner and Bateman regarding destructive CSF pressure/volume/plateau/B-waves and neurodegenerative diseases.
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Re: CCSVI and CCVBP

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Med Hypotheses. 2004;63(5):827-33.
Unifying concept for Alzheimer's disease, vascular dementia and normal pressure hydrocephalus - a hypothesis.Chakravarty A.

Abstract

The three common forms of dementias in the elderly include Alzheimer's disease (AD), vascular dementia (VD) and normal pressure hydrocephalus (NPH). These disorders are distinguished by their specific pathological features. However, overlapping clinical and imaging features in a given case are not too uncommon. Based on alterations in CSF dynamics study, a unifying concept in the pathogenesis of AD and NPH has been proposed recently which may have therapeutic implications. Altered CSF dynamics by affecting the absorptive process may lead to hydrocephalic change. This may also affect clearance of amyloid protein leading to increased amyloid deposition in brain parenchyma resulting in AD pathology. Hence it is likely that a subgroup of patients may have an AD-NPH syndrome who may be benefitted by CSF drainage procedure. The present author attempts to extend this concept to hypothesise a unifying concept to explain the pathophysiology of all the three disorders which may explain overlapping features observed clinically and in neuroimaging studies. It is surmised that altered CSF dynamics and hypoperfusion from vascular disease may be interlinked. The defective clearance of amyloid may also lead to amyloid angiopathy perpetuating hypoperfusion. Hypoperfusion may also affect formation as well as absorption of CSF altering clearance of amyloid and promoting vascular and parenchymal deposition. Thus the pathologies of AD, VaD and NPH get interrelated.
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NZer1
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Re: CCSVI and CCVBP

Post by NZer1 »

Hi Dr,
I have a change in symptoms that has finally been identified as 'going crosseyed'. My eyes are trying to focus in a horizontal context in one of the tests and keep on going in and out of focus because my eye sight has also deteriorated in the same time frame. Is there any data or do you have any input on this symptom developing the range of diseases that we talk about? Does it has a CSF/vascular issue essence?

I am still on the ABx protocol for CPn (2.5 years) and although it is very difficult to 'measure' what changes are occurring I can say that the way I am progressing is common for others who have gone before me with this treatment and they have had good benefits after 3-5 years and stopped the progression of what ever the baseline issue is. My 'MS' progression is still occurring as before. It may or may not be specifically CPn intracellular bacteria, it may be another 'type or group' and it may be a combination of body regions that are infected, such as gut and brain.

The whole unknown of post Lyme type of scenarios, or Lyme and co-infections, or DNA modified immune systems, or VDR blocking is near impossible to 'state' that 'the cause' is xyz, BUT the outcome is better than the pretreatment disabilities! :)

Regards,
Nigel
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