was hoping the ccsvi would reduce the swelling in feet/ankle
was hoping the ccsvi would reduce the swelling in feet/ankle
i thought blood flow was partially to blame for the swelling I have---but there is no difference-----altho my blood presure has stabalized from the wild swings it had before from 120-160---so now I am diving into the meds I take to see if they are the culpruit---- I am on the glutonfree/dairy free/ sugar free diet and do sleep inclined 4". and yes I do have those awful compression stockings-----anyone have the magic answer?
With my first CCSVI procedure the swelling in my left foot (that was so bad I was unable to put a shoe on) was gone, 5 hours after the procedure. I restenosed and the swelling returned. Again it disappeared after the second procedure and I restenosed and my swelling returned. It has been a year since my second procedure. I have tried 3 more times but my veins are so badly scarred, there is nothing the doctors can do. But what is strange, twice in the last year the swelling has disappeared, only for about 5 days. There is no explantion for this. I did not do anything or take anything different.
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blossom
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ccsvi
no magic answer. after the procedure my feet were still swelling like a balloon. but after going to the chiro. recommended by dr. flnagan to get my spine which is a mess, as good as i could get it, my feet do not swell nearly as bad. they are still purple except when in bed. i just started nucca not long ago and no changes but my case is very difficult.
NO---ILIAC WAS NOT LISTED AS WHAT HE DID---I AM JUST NOW STARTING TO HEAR ABOUT THIS VEIN---------I DON'T EVEN KNOW WHERE THAT IS------MCGUCKIN DID MY CCSVI IN MARCH--------I THINK THEY ARE LEARNING TONS EVERY DAY------MY THOUGHT IS TO GO BACK LATE IN THE YEAR HOPING THEY WILL LEARN A LOT UNTIL THEN---------------
Dr. McGuckin checks the iliac. Here's the youtube of him explaining what he does: www.youtube.com/watch?v=ogr1hAeKm1g
here's the nutritional spin on one route to problems with edema. i find it interesting that it highlights vitamin B1, which is one of the key nutrients included in the klenner protocol for MS, which specifies B1 supplements on the order of 1.2 grams per day if i remember correctly.
"In 1964 Myron Brin published a classic analysis of the five stages in the development of a vitamin or nutrient deficiency. He illustrated the schema with reference to vitamin B1 (thiamin).
In the first, or preliminary stage, inadequate B1 availability due to faulty diet, malabsorption or abnormal metabolism leads to a greatly reduced urinary B1 loss.
In the second, or biochemical stage, the activity of a key enzyme—transketolase—which is activated by B1, is significantly reduced. Adding B1 to a blood sample from a person at this stage increases his or her transketolase activity.
In the third, or physiologic stage, various general symptoms develop, such as lessened appetite, insomnia, increased irritability, and malaise.
In the fourth, or clinical stage, a constellation of symptoms classically specific to B1 deficiency disease (beri-beri) develops: e.g., intermittent claudication, polyneuritis, bradycardia, peripheral edema, and ophthalmoplegia (paralysis of eye muscles).
In the fifth, or anatomical stage, histopathological changes due to cellular structural damage are seen, such as cardiac hypertrophy, degeneration of the cerebellar granule layer, and swelling of the microglia.
Although Brin’s five-stage deficiency schema is exemplified with regard to B1, it is in principle applicable to any nutrient, as Brin himself notes. Brin’s schema is especially illuminating with regard to the RDAs, since the “just preventing failure of specific functions” and “just preventing specific deficiency signs” criteria of nutritional requirement, which is the basis of the RDA concept, are only evidenced in the fourth (clinical) and fifth (anatomical) stages of developing nutritional deficiency disease."
i just love that last paragraph
"In 1964 Myron Brin published a classic analysis of the five stages in the development of a vitamin or nutrient deficiency. He illustrated the schema with reference to vitamin B1 (thiamin).
In the first, or preliminary stage, inadequate B1 availability due to faulty diet, malabsorption or abnormal metabolism leads to a greatly reduced urinary B1 loss.
In the second, or biochemical stage, the activity of a key enzyme—transketolase—which is activated by B1, is significantly reduced. Adding B1 to a blood sample from a person at this stage increases his or her transketolase activity.
In the third, or physiologic stage, various general symptoms develop, such as lessened appetite, insomnia, increased irritability, and malaise.
In the fourth, or clinical stage, a constellation of symptoms classically specific to B1 deficiency disease (beri-beri) develops: e.g., intermittent claudication, polyneuritis, bradycardia, peripheral edema, and ophthalmoplegia (paralysis of eye muscles).
In the fifth, or anatomical stage, histopathological changes due to cellular structural damage are seen, such as cardiac hypertrophy, degeneration of the cerebellar granule layer, and swelling of the microglia.
Although Brin’s five-stage deficiency schema is exemplified with regard to B1, it is in principle applicable to any nutrient, as Brin himself notes. Brin’s schema is especially illuminating with regard to the RDAs, since the “just preventing failure of specific functions” and “just preventing specific deficiency signs” criteria of nutritional requirement, which is the basis of the RDA concept, are only evidenced in the fourth (clinical) and fifth (anatomical) stages of developing nutritional deficiency disease."
i just love that last paragraph
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